A mathematical model of prostate tumor growth under hormone therapy with mutation inhibitor

DOI10.1007/S00332-009-9056-ZMaRDI QIDQ5962228zbMATH OpenOpenAlexFDO

Authors Youshan Tao, Qian Guo, Kazuyuki Aihara

Publication date 21 September 2010

Published in Journal of Nonlinear Science (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1007/s00332-009-9056-z

zbMATH Keywords

free-boundary problempartial differential equationsprostate cancerhormone therapyinhibitors

Mathematics Subject Classification ID

Medical applications (general) (92C50) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Free boundary problems for PDEs (35R35) Developmental biology, pattern formation (92C15)

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Cites work

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• A mathematical model of intermittent androgen suppression for prostate cancer
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• A mathematical model to study the effects of drug resistance and vasculature on the response of solid tumors to chemotherapy
• A model at the macroscopic scale of prostate tumor growth under intermittent androgen suppression
• A nonlinear model with competition between prostate tumor cells and its application to intermittent androgen suppression therapy of prostate cancer
• A viscoelastic model for avascular tumor growth
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• MATHEMATICAL ANALYSIS AND CHALLENGES ARISING FROM MODELS OF TUMOR GROWTH
• MATHEMATICAL MODELING OF PROSTATE TUMOR GROWTH UNDER INTERMITTENT ANDROGEN SUPPRESSION WITH PARTIAL DIFFERENTIAL EQUATIONS
• MODELLING THE RESPONSE OF VASCULAR TUMOURS TO CHEMOTHERAPY: A MULTISCALE APPROACH
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• ON THE FOUNDATIONS OF CANCER MODELLING: SELECTED TOPICS, SPECULATIONS, AND PERSPECTIVES
• QUALITATIVE ANALYSIS OF A MEAN FIELD MODEL OF TUMOR-IMMUNE SYSTEM COMPETITION
• Vascular tumor growth and treatment: Consequences of polyclonality, competition and dynamic vascular support

Cited in

(19)

• Stability analysis on a hybrid PDE-ODE system describing intermittent hormonal therapy of prostate cancer
• Mathematical insights into neuroendocrine transdifferentiation of human prostate cancer cells
• Mathematical modelling for the role of CD\(4^+\)T cells in tumor-immune interactions
• A mathematical model of prostate tumor growth and androgen-independent relapse
• Optimal design of personalized prostate cancer therapy using infinitesimal perturbation analysis
• Androgen driven evolutionary population dynamics in prostate cancer growth
• A partial differential equation model of metastasized prostatic cancer
• A nonlinear competitive model of the prostate tumor growth under intermittent androgen suppression
• Practically scheduling hormone therapy for prostate cancer using a mathematical model
• A numerical simulation study of the dual role of \(5\alpha\)-reductase inhibitors on tumor growth in prostates enlarged by benign prostatic hyperplasia via stress relaxation and apoptosis upregulation
• Mathematical modelling of prostate cancer growth and its application to hormone therapy
• Dynamical aspects of a hybrid system describing intermittent androgen suppression therapy of prostate cancer
• Parameter estimation and sensitivity analysis for a model of tumor-immune interaction in the presence of immunotherapy and chemotherapy
• A new protocol for intermittent androgen suppression therapy of prostate cancer with unstable saddle-point dynamics
• A partial differential equation model and its reduction to an ordinary differential equation model for prostate tumor growth under intermittent hormone therapy
• Correlation between animal and mathematical models for prostate cancer progression
• Parameter estimation and optimal scheduling algorithm for a mathematical model of intermittent androgen suppression therapy for prostate cancer
• Numerical convergence and stability analysis for a nonlinear mathematical model of prostate cancer
• Combination of singularly perturbed vector field method and method of directly defining the inverse mapping applied to complex ODE system prostate cancer model

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