Modelling the role of tax expression in HTLV-I persistence in vivo
DOI10.1007/S11538-011-9657-1zbMATH Open1237.92031OpenAlexW2056909838WikidataQ51576794 ScholiaQ51576794MaRDI QIDQ417301FDOQ417301
Authors: Michael Y. Li, Aaron G. Lim
Publication date: 14 May 2012
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s11538-011-9657-1
Recommendations
- HTLV-I infection: A dynamic struggle between viral persistence and host immunity
- Modeling the T-cell dynamics and pathogenesis of HTLV-I infection
- Multistability in a model for CTL response to HTLV-I infection and its implications to HAM/TSP development and prevention
- Backward bifurcation in a model for HTLV-I infection of CD4\(^{+}\) T cells
- Population dynamics of HTLV-I infection: A discrete-time mathematical epidemic model approach
global stabilitybackward bifurcationlatently infected target cellspersistent viral infectionviral tax protein
Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60) Dynamical systems in biology (37N25) Probabilistic models, generic numerical methods in probability and statistics (65C20)
Cites Work
- Title not available (Why is that?)
- A model of HIV-1 pathogenesis that includes an intracellular delay
- Compound matrices and ordinary differential equations
- Backward bifurcation in a model for HTLV-I infection of CD4\(^{+}\) T cells
- Systems of Differential Equations Which Are Competitive or Cooperative: I. Limit Sets
- A Geometric Approach to Global-Stability Problems
- Additive compound matrices and an inequality for eigenvalues of symmetric stochastic matrices
- Title not available (Why is that?)
- Persistence in dynamical systems
- Global analysis of two tuberculosis models
- A criterion for stability of matrices
- A model for treatment strategy in the chemotherapy of AIDS
- Systems of Ordinary Differential Equations Which Generate an Order Preserving Flow. A Survey of Results
- Title not available (Why is that?)
Cited In (19)
- HTLV-I infection: A dynamic struggle between viral persistence and host immunity
- Mathematical modeling of HIV/HTLV co-infection with CTL-mediated immunity
- GLOBAL DYNAMICS OF A REACTION AND DIFFUSION MODEL FOR AN HTLV-I INFECTION WITH MITOTIC DIVISION OF ACTIVELY INFECTED CELLS
- Global dynamics of HIV/HTLV-I co-infection with effective CTL-mediated immune response
- Mathematical analysis of the global dynamics of a HTLV-I infection model, considering the role of cytotoxic T-lymphocytes
- The complete classification for global dynamics of a model for the persistence of HTLV-I infection
- Mathematical analysis of an HTLV-I infection model with the mitosis of CD4+ T cells and delayed CTL immune response
- Modeling and stability analysis of HIV/HTLV-I co-infection
- Analysis of an HTLV/HIV dual infection model with diffusion
- Stability of HIV/HTLV‐I co‐infection model with delays
- Global dynamics of an HTLV-1 model with cell-to-cell infection and mitosis
- Stability analysis of fuzzy HTLV-I infection model: a dynamic approach
- Global stability of HIV/HTLV co-infection model with CTL-mediated immunity
- Backward bifurcation of an HTLV-I model with immune response
- Stability analysis of general delayed HTLV-I dynamics model with mitosis and CTL immunity
- Modeling and analysis of a within-host HIV/HTLV-I co-infection
- Dynamic Scenario in HTLV-I Infection
- Title not available (Why is that?)
- Stability of HTLV/HIV dual infection model with mitosis and latency
This page was built for publication: Modelling the role of tax expression in HTLV-I persistence in vivo
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q417301)