scientific article; zbMATH DE number 1054972
Typical computational models of cardiac impulse propagation are based on simple, generic models that are usually solved on rectangular, isotropic domains. However, the intact ventricular myocardium differs from such models in three ways: the complex biochemical events in the cell membrane that govern the action potential, the microstructural basis of the communication of excitation, and the large-scale geometric complexity, fibrous architecture and spatial inhomogeneity of the whole heart. Sophisticated models of the membrane dynamics of the cardiac cell have been developed, as have models that explore the effects of the tissue microstructure on action potential propagation. However, the effects of the three-dimensional geometry, muscle fibre angle distribution, and inhomogeneity of the whole heart have remained largely unexplored, probably because of the lack of feasible computational methods to cope with such simulations. This chapter outlines a computational method developed to address this issue. The finite element method is a potentially powerful tool for physiological modelling -- in part because it allows the irregularities and inhomogeneities of biological systems to be included in the model description. For the entire collection see [Zbl 0867.92011].
- Development of finite element models for studying the electrical excitation of myocardium
- Modeling impulse propagation and extracellular potential distributions in anisotropic cardiac tissue using a finite volume element discretization
- scientific article; zbMATH DE number 1054973
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