PARAMETER DOMAINS FOR GENERATING SPATIAL PATTERN: A COMPARISON OF REACTION–DIFFUSION AND CELL-CHEMOTAXIS MODELS
From MaRDI portal
Publication:4374540
DOI10.1142/S0218127495001150zbMath0889.35051OpenAlexW2071803641MaRDI QIDQ4374540
Publication date: 4 February 1998
Published in: International Journal of Bifurcation and Chaos (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1142/s0218127495001150
Singular perturbations in context of PDEs (35B25) Reaction-diffusion equations (35K57) Kinetics in biochemical problems (pharmacokinetics, enzyme kinetics, etc.) (92C45) Developmental biology, pattern formation (92C15) Bifurcations in context of PDEs (35B32)
Related Items (14)
Lattice-Boltzmann model for bacterial chemotaxis ⋮ Parameter domains for spots and stripes in mechanical models for biological pattern formation ⋮ Economic agglomerations and spatio-temporal cycles in a spatial growth model with capital transport cost ⋮ Capital-induced labor migration in a spatial Solow model ⋮ Turing patterns with space varying diffusion coefficients: eigenfunctions satisfying the Legendre equation ⋮ A new necessary condition for Turing instabilities ⋮ Analysis of Lauffenburger-Kennedy bacterial infection model for tissue inflammation dynamics ⋮ An alternative smooth particle hydrodynamics formulation to simulate chemotaxis in porous media ⋮ Dynamics of Notch activity in a model of interacting signaling pathways ⋮ Transition of patterns in the cell-chemotaxis system with proliferation source ⋮ Overview of mathematical approaches used to model bacterial chemotaxis. II: Bacterial popu\-lations ⋮ Transitions and heteroclinic cycles in the general Gierer-Meinhardt equation and cardiovascular calcification model ⋮ Periodic pattern formation in the coupled chemotaxis-(Navier–)Stokes system with mixed nonhomogeneous boundary conditions ⋮ Transient dynamics and pattern formation: Reactivity is necessary for Turing instabilities
This page was built for publication: PARAMETER DOMAINS FOR GENERATING SPATIAL PATTERN: A COMPARISON OF REACTION–DIFFUSION AND CELL-CHEMOTAXIS MODELS