Activation thresholds in epidemic spreading with motile infectious agents on scale-free networks

DOI10.1063/1.5050807zbMATH Open1404.92203arXiv1808.01004OpenAlexW2885865335WikidataQ90838355 ScholiaQ90838355MaRDI QIDQ4644741FDOQ4644741

Authors: Diogo H. Silva, Silvio C. Ferreira 

Publication date: 8 January 2019

Published in: Chaos: An Interdisciplinary Journal of Nonlinear Science (Search for Journal in Brave)

Abstract: We investigate a fermionic susceptible-infected-susceptible model with mobility of infected individuals on uncorrelated scale-free networks with power-law degree distributions $P(k)sim{k}^{-gamma}$ of exponents $2<gamma<3$. Two diffusive processes with diffusion rate $D$ of an infected vertex are considered. In the extit{standard diffusion}, one of the nearest-neighbors is chosen with equal chance while in the extit{biased diffusion} this choice happens with probability proportional to the neighbor's degree. A non-monotonic dependence of the epidemic threshold on $D$ with an optimum diffusion rate $D_{a}st$, for which the epidemic spreading is more efficient, is found for standard diffusion while monotonic decays are observed in the biased case. The epidemic thresholds go to zero as the network size is increased and the form that this happens depends on the diffusion rule and degree exponent. We analytically investigated the dynamics using quenched and heterogeneous mean-field theories. The former presents, in general, a better performance for standard and the latter for biased diffusion models, indicating different activation mechanisms of the epidemic phases that are rationalized in terms of hubs or max $k$-core subgraphs.

Full work available at URL: https://arxiv.org/abs/1808.01004

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Mathematics Subject Classification ID

Epidemiology (92D30)

Cites Work

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Cited In (7)

• Epidemic spreading driven by biased random walks
• Modeling Communicable Diseases, Human Mobility, and Epidemics: A Review
• Epidemic spread in networks induced by deactivation mechanism
• Link importance assessment strategy based on improved \(k\)-core decomposition in complex networks
• Impacts of Game-Theoretic Activation on Epidemic Spread over Dynamical Networks
• Modelling the effect of heterogeneous vaccination on metapopulation epidemic dynamics
• Cascading failure of complex networks based on load redistribution and epidemic process

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