COEXISTENCE OF THREE OSCILLATORY MODES OF INSULIN SECRETION: MATHEMATICAL MODELING AND RELEVANCE TO GLUCOSE REGULATION

DOI10.1142/S0218339017500188zbMATH Open1373.92049OpenAlexW2617645362WikidataQ113777149 ScholiaQ113777149MaRDI QIDQ5348797FDOQ5348797

Authors: Hyuk Kang, Kyungreem Han, Segun Goh, M. Y. Choi 

Publication date: 21 August 2017

Published in: Journal of Biological Systems (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1142/s0218339017500188

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zbMATH Keywords

simulationsmodelingdiabetespancreatic \(\beta\)-cellsoscillatory insulin secretionwhole-body glucose regulation

Mathematics Subject Classification ID

Biochemistry, molecular biology (92C40) Systems biology, networks (92C42)

Cites Work

• Modeling the glucose-insulin regulatory system and ultradian insulin secretory oscillations with two explicit time delays
• An islet population model of the endocrine pancreas
• A simplified model for mitochondrial ATP production

Cited In (8)

• Diffusion induced oscillatory insulin secretion
• Beneficial effects of intercellular interactions between pancreatic islet cells in blood glucose regulation
• Lessons from models of pancreatic \(\beta \) cells for engineering glucose-sensing cells
• Modelling the effect of CSII on the control of glucose concentration in type 1 diabetes
• Hormonal effects on glucose regulation
• Integrated multiscale mathematical modeling of insulin secretion reveals the role of islet network integrity for proper oscillatory glucose-dose response
• A model for ultradian oscillations of insulin and glucose
• Mathematical modeling of insulin secretion and the role of glucose-dependent mobilization, docking, priming and fusion of insulin granules

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