Bifurcation for a free boundary problem modeling a small arterial plaque
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Publication:831091
DOI10.1016/J.JDE.2021.04.008zbMATH Open1466.92051OpenAlexW3156610091WikidataQ116009450 ScholiaQ116009450MaRDI QIDQ831091FDOQ831091
Abstract: Atherosclerosis, hardening of the arteries, originates from small plaque in the arteries; it is a major cause of disability and premature death in the United States and worldwide. In this paper, we study the bifurcation of a highly nonlinear and highly coupled PDE model describing the growth of arterial plaque in the early stage of atherosclerosis. The model involves LDL and HDL cholesterols, macrophage cells as well as foam cells, with the interface separating the plaque and blood flow regions being a free boundary. We establish finite branches of symmetry-breaking stationary solutions which bifurcate from the radially symmetric solution. Since plaque in reality is unlikely to be strictly radially symmetric, our result would be useful to explain the asymmetric shapes of plaque.
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Cited in
(12)- Symmetry-breaking longitude bifurcations for a free boundary problem modeling small plaques in three dimensions
- Periodic solution for a free boundary problem modeling small plaques
- A free boundary mathematical model of atherosclerosis
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- Bifurcation for a free-boundary problem modeling small plaques with reverse cholesterol transport
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