Early HIV infection in vivo: branching-process model for studying timing of immune reponses and drug therapy
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Publication:1579751
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Cites work
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- scientific article; zbMATH DE number 3337270 (Why is no real title available?)
- scientific article; zbMATH DE number 3349139 (Why is no real title available?)
- A theory for the age and generation time distribution of a microbial population
- Influence of delayed viral production on viral dynamics in HIV-1 infected patients
- Mathematical biology of HIV infections: Antigenic variation and diversity threshold
- On \(T\)-cell dynamics and the hyperactivation theory of AIDS pathogenesis
- Partial differential equations
- Random environments and stochastic calculus
- Two-event models for carcinogenesis: Incidence curves for childhood and adult tumors
Cited in
(16)- Early treatment gains for antibiotic administration and within human host time series data
- Sampling HIV intrahost genealogies based on a model of acute stage CTL response
- The probability of HIV infection in a new host and its reduction with microbicides
- A parameter sensitivity methodology in the context of HIV delay equation models
- Incorporation of variability into the modeling of viral delays in HIV infection dynamics
- Quantifying antiviral effects against simian/human immunodeficiency virus induced by host immune response
- An in-depth analysis of the HIV-1/AIDS dynamics by comprehensive mathematical modeling
- Stochastic analysis of pre- and postexposure prophylaxis against HIV infection
- A BRANCHING PROCESS MODEL OF DRUG RESISTANT HIV
- Probabilities of therapeutic extinction of HIV
- The stochastic dance of early HIV infection
- On simulating strongly-interacting, stochastic population models.
- On simulating strongly interacting, stochastic population models. II: Multiple compartments
- Stochastic investigation of HIV infection and the emergence of drug resistance
- Early HIV infection predictions: role of viral replication errors
- Modelling the impact of acute infection dynamics on the accumulation of HIV-1 mutations
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