Modelling the immune response to cancer: an individual-based approach accounting for the difference in movement between inactive and activated T cells
DOI10.1007/S11538-018-0412-8zbMATH Open1396.92037OpenAlexW2792202255WikidataQ88047750 ScholiaQ88047750MaRDI QIDQ1670474FDOQ1670474
Authors: Fiona R. Macfarlane, T. Lorenzi, M. A. J. Chaplain
Publication date: 5 September 2018
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: http://hdl.handle.net/10023/17262
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Processes with independent increments; Lévy processes (60G51) Applications of Markov chains and discrete-time Markov processes on general state spaces (social mobility, learning theory, industrial processes, etc.) (60J20) Medical applications (general) (92C50) Cell movement (chemotaxis, etc.) (92C17)
Cites Work
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- Recognition and learning in a mathematical model for immune response against cancer
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- Mathematical model of pulsed immunotherapy for superficial bladder cancer
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- In silico synergism and antagonism of an anti-tumour system intervened by coupling immunotherapy and chemotherapy: a mathematical modelling approach
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- Metamodeling the learning-hiding competition between tumours and the immune system: A kinematic approach
- Agent-based modeling of the context dependency in T cell recognition
- A dynamical model of tumour immunotherapy
- A preliminary mathematical model of skin dendritic cell trafficking and induction of T cell immunity
Cited In (10)
- A stochastic individual-based model to explore the role of spatial interactions and antigen recognition in the immune response against solid tumours
- Identifying and characterising the impact of excitability in a mathematical model of tumour-immune interactions
- A mathematical model to study the impact of intra-tumour heterogeneity on anti-tumour CD\(8^+\) T cell immune response
- Understanding the interplay of CAR-NK cells and triple-negative breast cancer: insights from computational modeling
- Mathematical modeling and bifurcation analysis for a biological mechanism of cancer drug resistance
- Macroscopic limit of a kinetic model describing the switch in T cell migration modes via binary interactions
- Mathematical modelling of cancer stem cell-targeted immunotherapy
- Extinction of bistable populations is affected by the shape of their initial spatial distribution
- Mathematical modeling of tumor-immune cell interactions
- A hybrid discrete-continuum modelling approach to explore the impact of T-cell infiltration on anti-tumour immune response
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