Mathematical model for the spread of drug resistance in \textit{Plasmodium falciparum} parasite considering transmission conditions

DOI10.1016/J.JTBI.2017.09.001MaRDI QIDQ1749042zbMATH OpenOpenAlexWikidataFDO

Authors Hernando Diaz, Andrés Olarte, Mario Morán Cañón

Publication date 15 May 2018

Published in Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/j.jtbi.2017.09.001

zbMATH Keywords

mathematical modeloptimal controlmalaria\textit{Plasmodium falciparum}drug resistencepyrimethamine

Mathematics Subject Classification ID

Medical applications (general) (92C50) Medical epidemiology (92C60) Epidemiology (92D30)

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Cites work

• scientific article; zbMATH DE number 3922442 (Why is no real title available?)
• scientific article; zbMATH DE number 1734466 (Why is no real title available?)
• An optimal control strategy to reduce the spread of malaria resistance
• Determining important parameters in the spread of malaria through the sensitivity analysis of a mathematical model

Cited in

(14)

• A mathematical model for antimalarial drug resistance
• A reaction-diffusion system modeling the spread of resistance to an antimalarial drug
• Intermittent preventive treatment (IPT) and the spread of drug resistant malaria
• Mathematical model for optimal use of sulfadoxine-pyrimethamine as a temporary malaria vaccine
• Mathematical model of susceptibility, resistance, and resilience in the within-host dynamics between a \textit{Plasmodium} parasite and the immune system
• The time distribution of sulfadoxine-pyrimethamine protection from malaria
• Multiple-strain malaria infection and its impacts on \textit{Plasmodium falciparum} resistance to antimalarial therapy: a mathematical modelling perspective
• A multiscale mathematical model of \textit{Plasmodium vivax} transmission
• The impact of heterogeneous transmission on the establishment and spread of antimalarial drug resistance
• A mathematical model of Malaria transmission dynamics in genetically resistant and susceptible population
• Transition from endemic behavior to eradication of malaria due to combined drug therapies: an agent-model approach
• A mechanistic model quantifies artemisinin-induced parasite growth retardation in blood-stage \textit{Plasmodium falciparum} infection
• A model for transmission of partial resistance to anti-malarial drugs
• Spread of antimalarial drug resistance in a population with superinfection

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