Mathematical model of intimal thickening in atherosclerosis: vessel stenosis as a free boundary problem
DOI10.1016/J.JTBI.2012.07.029zbMATH Open1397.92325OpenAlexW2071471339WikidataQ47248917 ScholiaQ47248917MaRDI QIDQ1790899FDOQ1790899
Authors: Pak-Wing Fok 
Publication date: 4 October 2018
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2012.07.029
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Medical applications (general) (92C50) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Free boundary problems for PDEs (35R35) Cell movement (chemotaxis, etc.) (92C17) Physiological flow (92C35)
Cites Work
- Mathematical and numerical modeling of early atherosclerotic lesions
- A mathematical model of atherogenesis as an inflammatory response
- An ODE model of early stages of atherosclerosis: mechanisms of the inflammatory response
- Long time evolution of atherosclerotic plaques
- Mathematical physiology. II: Systems physiology
- Growth of necrotic cores in atherosclerotic plaque
- On the mechanical stability of growing arteries
- Fibrin gel formation in a shear flow
- A nonlinear, anisotropic and axisymmetric model for balloon angioplasty
Cited In (9)
- A multiphase model of growth factor-regulated atherosclerotic cap formation
- Existence of solution of a free boundary problem for reaction-diffusion systems
- A biochemical and mechanical model of injury-induced intimal thickening
- Blood lipoproteins shape the phenotype and lipid content of early atherosclerotic lesion macrophages: a dual-structured mathematical model
- Stability analysis of mathematical modeling of atherosclerotic plaque formation
- Bifurcation and dynamics in a mathematical model of early atherosclerosis. How acute inflammation drives lesion development
- Well-posedness of a mathematical model of diabetic atherosclerosis with advanced glycation end-products
- A free boundary mathematical model of atherosclerosis
- A two-phase model of early fibrous cap formation in atherosclerosis
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