Long time evolution of atherosclerotic plaques
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Recommendations
- Mathematical modelling of the atherosclerotic plaque formation
- A spatially resolved and quantitative model of early atherosclerosis
- Bifurcation analysis of a model for atherosclerotic plaque evolution
- Stability analysis of mathematical modeling of atherosclerotic plaque formation
- Mathematical modeling and simulation of the evolution of plaques in blood vessels
Cites work
- A mathematical model of atherogenesis as an inflammatory response
- A new constitutive framework for arterial wall mechanics and a comparative study of material models
- A phenomenological model for atherosclerotic plaque growth and rupture
- An ODE model of early stages of atherosclerosis: mechanisms of the inflammatory response
- Atherosclerosis initiation modeled as an inflammatory process
- scientific article; zbMATH DE number 4070476 (Why is no real title available?)
- Lipoprotein oxidation and its significance for atherosclerosis: a mathematical approach
- Mathematical modelling of atherosclerosis as an inflammatory disease
- Mathematical modelling of the atherosclerotic plaque formation
Cited in
(30)- Modeling fibrous cap formation in atherosclerotic plaque development: stability and oscillatory behavior
- A quantitative model of early atherosclerotic plaques parameterized using in vitro experiments
- Mathematical model of intimal thickening in atherosclerosis: vessel stenosis as a free boundary problem
- A spatially resolved and quantitative model of early atherosclerosis
- Role of vascular smooth muscle cell phenotypic switching in plaque progression: a hybrid modeling study
- Optimal decision in a statistical process control with cubic loss
- A phenomenological model for atherosclerotic plaque growth and rupture
- A multiphase model of growth factor-regulated atherosclerotic cap formation
- Artificial stenoses for computational hemodynamics
- Statistical process control optimization with variable sampling interval and nonlinear expected loss
- A new correlation for inclusion of leaky junctions in macroscopic modeling of atherosclerotic lesion initiation
- Bifurcation analysis of a model for atherosclerotic plaque evolution
- Growth of necrotic cores in atherosclerotic plaque
- Studies on atherosclerotic plaque formation: a mathematical approach
- Optimal Time-Sampling in a Statistical Process Control with a Polynomial Expected Loss
- Stability analysis of mathematical modeling of atherosclerotic plaque formation
- Mathematical modelling of the effects of statins on the growth of necrotic core in atherosclerotic plaque
- A lipid-structured model of atherosclerotic plaque macrophages with lipid-dependent kinetics
- Nonnegative weak solutions for a mathematical model of atherosclerosis in the early stage
- Blood lipoproteins shape the phenotype and lipid content of early atherosclerotic lesion macrophages: a dual-structured mathematical model
- Mathematical modeling of atheroma plaque mechanobiology: distinguishing vulnerable versus non-vulnerable plaques
- Effects of hematocrit levels on hemodynamics and atherosclerosis risk in the left main coronary artery: a comparative computational mechanobiological study
- A two-phase model of early atherosclerotic plaque development with LDL toxicity effects
- A spatially resolved and lipid-structured model for macrophage populations in early human atherosclerotic lesions
- A mathematical model for atherosclerotic plaque formation and arterial wall remodelling
- Asymptotic stability for a free boundary model of an atherosclerotic plaque formation in the presence of reverse cholesterol transport with Holling type-III functional response
- Mathematical modeling and analysis of atherosclerosis based on fluid-multilayered poroelastic structure interaction model
- Long-term simulation of large deformation, mechano-chemical fluid-structure interactions in ALE and fully Eulerian coordinates
- Intestinal alkaline phosphatase interaction gut model
- Bifurcation and dynamics in a mathematical model of early atherosclerosis. How acute inflammation drives lesion development
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