Blood lipoproteins shape the phenotype and lipid content of early atherosclerotic lesion macrophages: a dual-structured mathematical model
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Publication:6601322
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Cites work
- A lipid-structured model of atherosclerotic plaque macrophages with lipid-dependent kinetics
- A mathematical model of atherosclerosis with reverse cholesterol transport and associated risk factors
- A multiphase model of growth factor-regulated atherosclerotic cap formation
- A new lipid-structured model to investigate the opposing effects of LDL and HDL on atherosclerotic plaque macrophages
- A phenotype-structured model to reproduce the avascular growth of a tumor and its interaction with the surrounding environment
- A quantitative model of early atherosclerotic plaques parameterized using in vitro experiments
- A spatially and size-structured population model with unbounded birth process
- A two-phase model of early fibrous cap formation in atherosclerosis
- Analysis of a population model structured by the cells molecular content
- Athero-protective effects of high density lipoproteins (HDL): an ODE model of the early stages of atherosclerosis
- Bifurcation and dynamics in a mathematical model of early atherosclerosis. How acute inflammation drives lesion development
- Growth of necrotic cores in atherosclerotic plaque
- Hopf bifurcation for a spatially and age structured population dynamics model
- Long time evolution of atherosclerotic plaques
- Low-density lipoprotein (LDL) transport in an artery -- a simplified analytical solution
- Macrophage anti-inflammatory behaviour in a multiphase model of atherosclerotic plaque development
- Mathematical model of intimal thickening in atherosclerosis: vessel stenosis as a free boundary problem
- Mathematical modelling of the atherosclerotic plaque formation
- Mathematical study of an inflammatory model for atherosclerosis: a nonlinear renewal equation
- Modeling of low-density lipoprotein (LDL) transport in the artery-effects of hypertension
- Modelling preferential phagocytosis in atherosclerosis: delineating timescales in plaque development
- Nonlinear physiologically structured population models with two internal variables
- Propagation dynamics for an age-structured population model in time-space periodic habitat
- Spatial-temporal patterns of a two age structured population model with spatial non-locality
- Spatio-genetic and phenotypic modelling elucidates resistance and re-sensitisation to treatment in heterogeneous melanoma
- Spatio-temporal modelling of phenotypic heterogeneity in tumour tissues and its impact on radiotherapy treatment
- Threshold behaviour of a SI epidemiological model with two structuring variables
- Well-posedness of a mathematical model of diabetic atherosclerosis
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