A DNA-structured mathematical model of cell-cycle progression in cyclic hypoxia
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Publication:2146758
DOI10.1016/J.JTBI.2022.111104zbMATH Open1491.92047OpenAlexW4220798524WikidataQ113869613 ScholiaQ113869613MaRDI QIDQ2146758FDOQ2146758
Authors: Giulia L. Celora, Samuel B. Bader, Ester M. Hammond, Joe Pitt-Francis, Philip K. Maini, H. M. Byrne 
Publication date: 21 June 2022
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2022.111104
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Cites Work
- A nonlinear structured population model of tumor growth with quiescence
- Modelling the flow of cytometric data obtained from unperturbed human tumour cell lines: parameter fitting and comparison
- A nonlinear model of age and size-structured populations with applications to cell cycles
- A mathematical model for analysis of the cell cycle in cell lines derived from human tumors
- A generalised age- and phase-structured model of human tumour cell populations both unperturbed and exposed to a range of cancer therapies
- An age structured cell cycle model with crowding
- A mathematical model of the effects of hypoxia on the cell-cycle of normal and cancer cells
- A mathematical model of cell cycle progression applied to the MCF-7 breast cancer cell line
- AN IN VITRO CELL POPULATION DYNAMICS MODEL INCORPORATING CELL SIZE, QUIESCENCE, AND CONTACT INHIBITION
- The steady-states of a multi-compartment, age–size distribution model of cell-growth
- A novel mathematical model of heterogeneous cell proliferation
- Modelling the balance between quiescence and cell death in normal and tumour cell populations
- Modelling cell death in human tumour cell lines exposed to the anticancer drug paclitaxel
- Bayesian calibration, validation and uncertainty quantification for predictive modelling of tumour growth: a tutorial
- Unraveling the complexity of cell cycle effects of anticancer drugs in cell populations
- Variability in the timing of G\(_{1}\)/S transition
- A mathematical model for cell cycle-specific cancer virotherapy
Cited In (5)
- Mathematical Modelling of the Cell Cycle Stress Response
- Characterising cancer cell responses to cyclic hypoxia using mathematical modelling
- A mathematical model of the effects of hypoxia on the cell-cycle of normal and cancer cells
- Quiescence as a mechanism for cyclical hypoxia and acidosis
- A mathematical model of HiF-\(1{\alpha}\)-mediated response to hypoxia on the G1/S transition
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