Complex bursting in pancreatic islets: a potential glycolytic mechanism
From MaRDI portal
Publication:2187665
DOI10.1016/J.JTBI.2004.02.022zbMATH Open1439.92106OpenAlexW2029995008WikidataQ80161650 ScholiaQ80161650MaRDI QIDQ2187665FDOQ2187665
Authors: Yanyan Li
Publication date: 3 June 2020
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2004.02.022
Recommendations
- Phantom bursting may underlie electrical bursting in single pancreatic \(\beta\)-cells
- Electrical activity and insulin release in pancreatic beta cells
- Transitions between bursting modes in the integrated oscillator model for pancreatic \(\beta\)-cells
- Phantom bursting is highly sensitive to noise and unlikely to account for slow bursting in \(\beta\)-cells: considerations in favor of metabolically driven oscillations
- A calcium-based phantom bursting model for pancreatic islets
Physiology (general) (92C30) Kinetics in biochemical problems (pharmacokinetics, enzyme kinetics, etc.) (92C45)
Cites Work
Cited In (3)
Uses Software
This page was built for publication: Complex bursting in pancreatic islets: a potential glycolytic mechanism
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q2187665)