Mathematical modeling of glioma invasion: acid- and vasculature mediated go-or-grow dichotomy and the influence of tissue anisotropy
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Publication:2243281
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Cites work
- `Go or grow': the key to the emergence of invasion in tumour progression?
- A general reaction-diffusion model of acidity in cancer invasion
- A hybrid model for cell proliferation and migration in glioblastoma
- A mathematical model for pattern formation of glioma cells outside the tumor spheroid core
- A mathematical model of tumour and blood pHe regulation: the \(\mathrm{HCO}^-_3/\mathrm{CO}_2\) buffering system
- A multiscale approach to cell migration in tissue networks
- A multiscale model for glioma spread including cell-tissue interactions and proliferation
- A multiscale model for pH-tactic invasion with time-varying carrying capacities
- A multiscale modeling approach to glioma invasion with therapy
- A patient-specific anisotropic diffusion model for brain tumour spread
- A pharmacologically based multiscale mathematical model of angiogenesis and its use in investigating the efficacy of a new cancer treatment strategy
- A spatial model of tumor-host interaction: application of chemotherapy
- A stochastic model featuring acid-induced gaps during tumor progression
- A stochastic multiscale model for acid mediated cancer invasion
- A strongly degenerate diffusion-haptotaxis model of tumour invasion under the go-or-grow dichotomy hypothesis
- An image-driven parameter estimation problem for a reaction-diffusion glioma growth model with mass effects
- Complexity and mathematical tools toward the modelling of multicellular growing systems
- Density-dependent quiescence in glioma invasion: instability in a simple reaction–diffusion model for the migration/proliferation dichotomy
- Derivation of the bacterial run-and-tumble kinetic equation from a model with biochemical pathway
- Effective equations for anisotropic glioma spread with proliferation: a multiscale approach and comparisons with previous settings
- Glioma follow white matter tracts: a multiscale DTI-based model
- Glioma invasion and its interplay with nervous tissue and therapy: a multiscale model
- Global existence for a go-or-grow multiscale model for tumor invasion with therapy
- Higher-order models for glioma invasion: from a two-scale description to effective equations for mass density and momentum
- scientific article; zbMATH DE number 42093 (Why is no real title available?)
- Hypoxic cell waves around necrotic cores in glioblastoma: a biomathematical model and its therapeutic implications
- Investigation of the migration/proliferation dichotomy and its impact on avascular glioma invasion
- Mathematical modelling of glioma growth: the use of diffusion tensor imaging (DTI) data to predict the anisotropic pathways of cancer invasion
- Mathematical modelling of tumour acidity
- Modeling complex living systems. A kinetic theory and stochastic game approach.
- Modeling glioma invasion with anisotropy- and hypoxia-triggered motility enhancement: from subcellular dynamics to macroscopic PDEs with multiple taxis
- Modeling multiple taxis: tumor invasion with phenotypic heterogeneity, haptotaxis, and unilateral interspecies repellence
- Multiscale modeling of glioma pseudopalisades: contributions from the tumor microenvironment
- On a class of multiscale cancer cell migration models: well-posedness in less regular function spaces
- On a coupled SDE-PDE system modeling acid-mediated tumor invasion
- On a structured multiscale model for acid-mediated tumor invasion: the effects of adhesion and proliferation
- On some models for cancer cell migration through tissue networks
- The diffusion limit of transport equations. II: Chemotaxis equations
Cited in
(33)- Deterministic and stochastic modeling for PDGF-driven gliomas reveals a classification of gliomas
- Multiphase modelling of glioma pseudopalisading under acidosis
- Gibbs point field model quantifies disorder in microvasculature of U87-glioblastoma
- Viability in a non-local population model structured by size and spatial position
- Mathematical analysis of a model for proliferative-to-invasive transition of hypoxic glioma cells
- Glioma invasion and its interplay with nervous tissue and therapy: a multiscale model
- Mathematical modelling of glioma growth: the use of diffusion tensor imaging (DTI) data to predict the anisotropic pathways of cancer invasion
- Traveling wave speed and profile of a ``go or grow glioblastoma multiforme model
- Investigation of the migration/proliferation dichotomy and its impact on avascular glioma invasion
- Higher-order models for glioma invasion: from a two-scale description to effective equations for mass density and momentum
- A Novel Derivation of Rigorous Macroscopic Limits from a Micro-Meso Description of Signal-Triggered Cell Migration in Fibrous Environments
- A glioblastoma PDE-ODE model including chemotaxis and vasculature
- Multiscale modeling of glioma invasion: from receptor binding to flux-limited macroscopic PDEs
- Modeling glioma invasion with anisotropy- and hypoxia-triggered motility enhancement: from subcellular dynamics to macroscopic PDEs with multiple taxis
- A mathematical model of dynamic glioma–host interactions: receptor-mediated invasion and local proteolysis
- A stochastic hierarchical model for low grade glioma evolution
- Modelling microtube driven invasion of glioma
- On a mathematical model for cancer invasion with repellent pH-taxis and nonlocal intraspecific interaction
- Data driven modeling of pseudopalisade pattern formation
- Mathematical modeling of glioma invasion and therapy approaches via kinetic theory of active particles
- An imaging-informed mechanical framework to provide a quantitative description of brain tumour growth and the subsequent deformation of white matter tracts
- Phenotypic switching mechanisms determine the structure of cell migration into extracellular matrix under the `go-or-grow' hypothesis
- Modelling and simulation of anisotropic growth in brain tumours through poroelasticity: a study of ventricular compression and therapeutic protocols
- Calcium signalling in glioblastoma networks of different topologies and possible treatments
- New trends in kinetic theory toward the complexity of living systems
- Go-or-grow models in biology: a monster on a leash
- On a mathematical model for tissue regeneration
- Multi-scale modeling of Snail-mediated response to hypoxia in tumor progression
- Derivation and travelling wave analysis of phenotype-structured haptotaxis models of cancer invasion
- Mathematical modeling and analysis of a tumor invasion problem with angiogenesis and taxis cascade
- Co-evolution of viruses and games
- An in-silico approach to meniscus tissue regeneration: modeling, numerical simulation, and experimental analysis
- A multiscale model for glioma spread including cell-tissue interactions and proliferation
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