Model of influenza A virus infection: dynamics of viral antagonism and innate immune response

DOI10.1016/J.JTBI.2014.02.029MaRDI QIDQ2632814zbMATH OpenOpenAlexWikidataFDO

Authors Yanyan Li

Publication date 15 May 2019

Published in Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL http://europepmc.org/articles/pmc4012420

zbMATH Keywords

immunologyinterferonNS1ODE model

Mathematics Subject Classification ID

Epidemiology (92D30)

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Cites work

• Multi-objective optimization using evolutionary algorithms

Cited in

(20)

• Optimal control of acute myeloid leukaemia
• A model for influenza with vaccination and antiviral treatment
• Spatio-temporal dynamics of host-virus competition: a model study of influenza A
• Modelling cross-reactivity and memory in the cellular adaptive immune response to influenza infection in the host
• Compartmental model suggests importance of innate immune response to COVID-19 infection in rhesus macaques
• Mathematical modelling of OAS2 activation by dsRNA and effects of dsRNA lengths
• Age-related changes to the immune system exacerbate the inflammatory response to pandemic H1N1 infection
• A dynamical model of human immune response to influenza A virus infection
• Modelling within-host macrophage dynamics in influenza virus infection
• Mathematical modelling of acute virus influenza A infections
• Dynamics of influenza virus and human host interactions during infection and replication cycle
• Influenza A virus antigen and neutralizing antibody mechanism
• Models of cytokine dynamics in the inflammatory response of viral zoonotic infectious diseases
• Ex vivo experiments shed light on the innate immune response from influenza virus
• An in-host model of acute infection: measles as a case study
• Different routes of infection of H5N1 lead to changes in infecting time
• A target-cell limited model can reproduce influenza infection dynamics in hosts with differing immune responses
• A within-host mathematical model of H9N2 avian influenza infection and type-I interferon response pathways in chickens
• Mechanistic modeling explains the dsRNA length-dependent activation of the RIG-I mediated immune response
• Modeling the interaction of cytotoxic T lymphocytes and influenza virus infected epithelial cells

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