Understanding PGE2, LXA4 and LTB4 balance during \textit{Mycobacterium tuberculosis} infection through mathematical model
DOI10.1016/J.JTBI.2015.10.025zbMATH Open1343.92284OpenAlexW2115082807WikidataQ40917266 ScholiaQ40917266MaRDI QIDQ304757FDOQ304757
Authors: Gabriele Pedruzzi, Phonindra Nath Das, Kanury VS. Rao, S. Chatterjee 
Publication date: 26 August 2016
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2015.10.025
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differential equations\(LXA_4:LTB_4\)\(PGE_2\)\textit{Mycobacterium tuberculosis}host-pathogen interaction
Cites Work
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- An alternative way to compute Fourier amplitude sensitivity test (FAST).
- The timing of TNF and IFN-\(\gamma\) signaling affects macrophage activation strategies during \textit{Mycobacterium tuberculosis} infection
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Cited In (7)
- Deciphering the virulence of \textit{Mycobacterium avium} subsp. \textit{paratuberculosis} isolates in animal macrophages using mathematical models
- Deterministic and stochastic in-host tuberculosis models for bacterium-directed and host-directed therapy combination
- An investigation of tuberculosis progression revealing the role of macrophages apoptosis via sensitivity and bifurcation analysis
- Mathematical model of mycobacterium-host interaction describes physiology of persistence
- A simple in-host model for \textit{Mycobacterium tuberculosis} that captures all infection outcomes
- Restoration of cytosolic calcium inhibits \textit{Mycobacterium tuberculosis} intracellular growth: theoretical evidence and experimental observation
- Critical observation of WHO recommended multidrug therapy on the disease leprosy through mathematical study
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