An HIV model with age-structured latently infected cells
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Publication:3300917
DOI10.1080/17513758.2016.1198835zbMATH Open1447.92191OpenAlexW2464926540WikidataQ40638814 ScholiaQ40638814MaRDI QIDQ3300917FDOQ3300917
Authors: Areej Alshorman, Chathuri Samarasinghe, Wenlian Lu, Libin Rong 
Publication date: 31 July 2020
Published in: Journal of Biological Dynamics (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1080/17513758.2016.1198835
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Medical applications (general) (92C50) Stability of solutions to ordinary differential equations (34D20)
Cites Work
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- Emergence of HIV-1 drug resistance during antiretroviral treatment
- HIV-1 infection and low steady state viral loads
- The effects of latent infection on the dynamics of HIV
- Modeling HIV persistence, the latent reservoir, and viral blips
- A stage structured predator-prey model and its dependence on maturation delay and death rate
- How May Infection-Age-Dependent Infectivity Affect the Dynamics of HIV/AIDS?
- Title not available (Why is that?)
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Mathematical Analysis of Age‐Structured HIV‐1 Dynamics with Combination Antiretroviral Therapy
- Stochastic population switch may explain the latent reservoir stability and intermittent viral blips in HIV patients on suppressive therapy
- Stochastic modelling of viral blips in HIV-1-infected patients: effects of inhomogeneous density fluctuations
- Global stability of an infection-age structured HIV-1 model linking within-host and between-host dynamics
- Immuno-modulatory strategies for reduction of HIV reservoir cells
- Conditions for transient viremia in deterministic in-host models: viral blips need no exogenous trigger
- A Two-Strain Tuberculosis Model with Age of Infection
- Latently infected cell activation: a way to reduce the size of the HIV reservoir?
- Influence of raltegravir intensification on viral load and 2-LTR dynamics in HIV patients on suppressive antiretroviral therapy
- Mathematical analysis of multiscale models for hepatitis C virus dynamics under therapy with direct-acting antiviral agents
Cited In (17)
- Stability analysis of an age-structured viral infection model with latency
- Analysis of HIV latent infection model with multiple infection stages and different drug classes
- Periodic solutions of an infected-age structured HIV model with the latent factor and different transmission modes
- Threshold dynamics of a diffusive HIV infection model with infection-age, latency and cell-cell transmission
- Global dynamics of an age-structured HIV infection model incorporating latency and cell-to-cell transmission
- Global dynamics of an age-structured model for HIV viral dynamics with latently infected T cells
- A new model describing the development of memory/latency during HIV infection
- Dynamical analysis of a nonlocal delayed and diffusive HIV latent infection model with spatial heterogeneity
- Mathematical analysis of an age-structured HIV-1 infection model with CTL immune response
- Analysis of an HIV infection model incorporating latency age and infection age
- A study of latency, reactivation and apoptosis throughout HIV pathogenesis
- An age-structured within-host HIV model with T-cell competition
- Dynamics of an age-structured HIV model with super-infection
- A note on the global properties of an age-structured viral dynamic model with multiple target cell populations
- Numerical analysis of age-structured HIV model with general transmission mechanism
- An age-structured model of HIV latent infection with two transmission routes: analysis and optimal control
- Persistence of low levels of plasma viremia and of the latent reservoir in patients under ART: a fractional-order approach
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