Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
From MaRDI portal
Publication:1013286
DOI10.1016/J.MBS.2008.10.006zbMATH Open1158.92028OpenAlexW2039370209WikidataQ37133236 ScholiaQ37133236MaRDI QIDQ1013286FDOQ1013286
Authors: Libin Rong, Alan S. Perelson 
Publication date: 17 April 2009
Published in: Mathematical Biosciences (Search for Journal in Brave)
Full work available at URL: http://europepmc.org/articles/pmc2657607
Recommendations
- Stochastic population switch may explain the latent reservoir stability and intermittent viral blips in HIV patients on suppressive therapy
- Modeling HIV persistence, the latent reservoir, and viral blips
- A new model describing the development of memory/latency during HIV infection
- Modeling the long-term control of viremia in HIV-1 infected patients treated with antiretroviral therapy.
- The distribution of viral blips observed in HIV-1 infected patients treated with combination antiretroviral therapy
Cites Work
- Dynamics of HIV infection of CD4\(^ +\) T cells
- HIV-1 infection and low steady state viral loads
- The distribution of viral blips observed in HIV-1 infected patients treated with combination antiretroviral therapy
- Duration of an intermittent episode of viremia
- Opportunistic infection as a cause of transient viremia in chronically infected HIV patients under treatment with HAART
- Title not available (Why is that?)
- Modeling the long-term control of viremia in HIV-1 infected patients treated with antiretroviral therapy.
- Effects of asymmetric division on a stochastic model of the cell division cycle
- Effect of stem cell turnover rates on protection against cancer and aging
Cited In (61)
- Influence of raltegravir intensification on viral load and 2-LTR dynamics in HIV patients on suppressive antiretroviral therapy
- Dynamics and asymptotical profiles of an age-structured viral infection model with spatial diffusion
- A stochastic HIV infection model with latent infection and antiretroviral therapy
- Optimal control in HIV chemotherapy with termination viral load and latent reservoir
- A model of HIV-1 infection with two time delays: mathematical analysis and comparison with patient data
- Impact of latently infected cells on strain archiving within HIV hosts
- Analysis of HIV latent infection model with multiple infection stages and different drug classes
- Dissociation of HIV-1 from follicular dendritic cells during HAART: Mathematical analysis
- Modeling pharmacodynamics on HIV latent infection: choice of drugs is key to successful cure via early therapy
- Analysis and optimal control of an HIV model with logistic growth and infected cells in eclipse phase
- Asymptotical profiles of a viral infection model with multi-target cells and spatial diffusion
- Dynamics of an HIV model with multiple infection stages and treatment with different drug classes
- Dynamical analysis of antigen-driven t-cell infection model with multiple delays
- Dynamics of an HIV model with cytotoxic T-lymphocyte memory
- Global dynamics of a latent HIV infection model with general incidence function and multiple delays
- Analysis of an HIV model with post-treatment control
- Andronov-Hopf bifurcation and sensitivity analysis of a time-delay HIV model with logistic growth and antiretroviral treatment
- Mathematical analysis of an HIV latent infection model including both virus-to-cell infection and cell-to-cell transmission
- HIV infection dynamics and viral rebound: modeling results from humanized mice
- Global properties for a class of latent HIV infection dynamics model with CTL immune response
- Stochastic population switch may explain the latent reservoir stability and intermittent viral blips in HIV patients on suppressive therapy
- Effects of eclipse phase and delay on the dynamics of HIV infection
- Modeling HIV persistence, the latent reservoir, and viral blips
- Stochastic modelling of viral blips in HIV-1-infected patients: effects of inhomogeneous density fluctuations
- Stability and Hopf bifurcation in a delayed HIV infection model with general incidence rate and immune impairment
- Mathematical analysis of a class of HIV infection models of \(\mathrm{CD4}^{+}\) T-cells with combined antiretroviral therapy
- Dynamics of a new HIV model with the activation status of infected cells
- An HIV model with age-structured latently infected cells
- The effects of CTL immune response on HIV infection model with potent therapy, latently infected cells and cell-to-cell viral transmission
- Studying HIV latency by modeling the interaction between HIV proteins and the innate immune response
- Dynamics of an HIV infection model with virus diffusion and latently infected cell activation
- Modeling within-host viral dynamics: the role of CTL immune responses in the evolution of drug resistance
- Qualitative analysis of a generalized virus dynamics model with both modes of transmission and distributed delays
- Immuno-modulatory strategies for reduction of HIV reservoir cells
- Backward bifurcations, turning points and rich dynamics in simple disease models
- Analysis of HIV models with two time delays
- Spatiotemporal dynamics for a diffusive HIV-1 infection model with distributed delays and CTL immune response
- Analysis of an HIV infection model incorporating latency age and infection age
- Spatial and temporal dynamics of a viral infection model with two nonlocal effects
- Stochastic HIV model coupled with pharmacokinetics and drug adherence may explain intermittent viral blips
- Viral dynamics of an HIV model with latent infection incorporating antiretroviral therapy
- Latently infected cell activation: a way to reduce the size of the HIV reservoir?
- Stochastic modeling and simulation of viral evolution
- Towards a new combination therapy with vectored immunoprophylaxis for HIV: modeling ``Shock and kill strategy
- Modeling the role of macrophages in HIV persistence during antiretroviral therapy
- Global exponential stability of a delayed HIV infection model with a nonlinear incidence rate
- Andronov-Hopf and Neimark-Sacker bifurcations in time-delay models of HIV transmission
- Andronov-Hopf and Neimark-Sacker bifurcations in time-delay differential equations and difference equations with applications to models for diseases and animal populations
- A delay model for persistent viral infections in replicating cells
- Bistability analysis of an HIV model with immune response
- Conflict and accord of optimal treatment strategies for HIV infection within and between hosts
- Mathematical analysis of an HIV model with latent reservoir, delayed CTL immune response and immune impairment
- Global dynamics and cost-effectiveness analysis of HIV pre-exposure prophylaxis and structured treatment interruptions based on a multi-scale model
- Dynamics of the HIV infection under antiretroviral therapy: a cellular automata approach
- Bistability of an HIV model with immune impairment
- The effects of delay and impulsive drug therapy in an HIV model with CTLs immune response
- A new model describing the development of memory/latency during HIV infection
- The SEIR model with pulse and diffusion of virus in the environment
- Ongoing HIV replication in lymph node sanctuary sites in treated individuals contributes to the total latent HIV at a very slow rate
- Stability and Hopf bifurcation of HIV-1 model with Holling II infection rate and immune delay
- Dynamics analysis of an HIV infection model with latent reservoir, delayed CTL immune response and immune impairment
This page was built for publication: Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q1013286)
