The effects of CTL immune response on HIV infection model with potent therapy, latently infected cells and cell-to-cell viral transmission
DOI10.3934/MBE.2019341zbMATH Open1470.92074OpenAlexW2965881293WikidataQ91167868 ScholiaQ91167868MaRDI QIDQ2045491FDOQ2045491
Authors: Ting Guo, Zhipeng Qiu
Publication date: 13 August 2021
Published in: Mathematical Biosciences and Engineering (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.3934/mbe.2019341
Recommendations
- Threshold dynamics of HIV-1 virus model with cell-to-cell transmission, cell-mediated immune responses and distributed delay
- Stability of a general CTL-mediated immunity HIV infection model with silent infected cell-to-cell spread
- Stability of HIV-1 infection with saturated virus-target and infected-target incidences and CTL immune response
- Mathematical analysis of the dynamics of HIV infection with CTL immune response and cure rate
- Mathematical analysis of an HIV model with latent reservoir, delayed CTL immune response and immune impairment
HIVantiretroviral therapyglobal stabilityCTL immune responselatently infected cellscell-to-cell viral transmission
Medical epidemiology (92C60) Epidemiology (92D30) Global stability of solutions to ordinary differential equations (34D23) Pathology, pathophysiology (92C32)
Cites Work
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Dynamics of HIV infection of CD4\(^ +\) T cells
- Emergence of HIV-1 drug resistance during antiretroviral treatment
- HIV-1 infection and low steady state viral loads
- Title not available (Why is that?)
- The effects of latent infection on the dynamics of HIV
- Modeling HIV persistence, the latent reservoir, and viral blips
- Global stability and Hopf bifurcation of an HIV-1 infection model with saturation incidence and delayed CTL immune response
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Threshold dynamics of HIV-1 virus model with cell-to-cell transmission, cell-mediated immune responses and distributed delay
- Global threshold dynamics in an HIV virus model with nonlinear infection rate and distributed invasion and production delays
- Global threshold dynamics in a five-dimensional virus model with cell-mediated, humoral immune responses and distributed delays
- Dynamics of an HIV model with multiple infection stages and treatment with different drug classes
- Global properties of a delayed HIV infection model with CTL immune response
- Global dynamics of a cell mediated immunity in viral infection models with distributed delays
- Stability and Hopf bifurcation of a HIV infection model with CTL-response delay
- Viral dynamics of an HIV model with latent infection incorporating antiretroviral therapy
- Viral dynamics model with CTL immune response incorporating antiretroviral therapy
- Global analysis of within host virus models with cell-to-cell viral transmission
- The role of backward mutations on the within-host dynamics of HIV-1
- Global stability of a delayed HIV infection model with nonlinear incidence rate
- Modelling two different therapy strategies for drug T-20 on HIV-1 patients
- Mathematical analysis of a class of HIV infection models of \(\mathrm{CD4}^{+}\) T-cells with combined antiretroviral therapy
- Optimal control of an HIV infection model with logistic growth, CTL immune response and infected cells in eclipse phase
- Influence of raltegravir intensification on viral load and 2-LTR dynamics in HIV patients on suppressive antiretroviral therapy
- Persistence of low levels of plasma viremia and of the latent reservoir in patients under ART: a fractional-order approach
Cited In (23)
- Modeling the effect of reactive oxygen species and CTL immune response on HIV dynamics
- Modeling the role of acquired immune response and antiretroviral therapy in the dynamics of HIV infection
- Stability of a general adaptive immunity HIV infection model with silent infected cell-to-cell spread
- HIV infection dynamics and viral rebound: modeling results from humanized mice
- Modeling and stability analysis of HIV/HTLV-I co-infection
- Analysis of an HTLV/HIV dual infection model with diffusion
- Stability of a general CTL-mediated immunity HIV infection model with silent infected cell-to-cell spread
- Dynamics of a new HIV model with the activation status of infected cells
- Stability of HIV/HTLV‐I co‐infection model with delays
- Modeling within-host viral dynamics: the role of CTL immune responses in the evolution of drug resistance
- Threshold dynamics of HIV-1 virus model with cell-to-cell transmission, cell-mediated immune responses and distributed delay
- Impact of gene-modified T cells on HIV infection dynamics
- Exploring the benefits of antibody immune response in HIV-1 infection using a discrete model
- An in-host HIV-1 infection model incorporating quiescent and activated \(\mathrm{CD}4^+\) T cells as well as CTL response
- Latently infected cell activation: a way to reduce the size of the HIV reservoir?
- Mathematical analysis of the dynamics of HIV infection with CTL immune response and cure rate
- Modeling virus-stimulated proliferation of CD \(4^+\) T-cell, cell-to-cell transmission and viral loss in HIV infection dynamics
- Modeling and analysis of a within-host HIV/HTLV-I co-infection
- Modeling HIV multiple infection
- Co-infection dynamics between HIV-HTLV-I disease with the effects of cytotoxic T-lymphocytes, saturated incidence rate and study of optimal control
- Saturation effects for CTL mediated control of HIV-1 infection: a mathematical study
- Dynamic analysis of a latent HIV infection model with CTL immune and antibody responses
- Stability of HTLV/HIV dual infection model with mitosis and latency
This page was built for publication: The effects of CTL immune response on HIV infection model with potent therapy, latently infected cells and cell-to-cell viral transmission
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q2045491)