The effects of CTL immune response on HIV infection model with potent therapy, latently infected cells and cell-to-cell viral transmission
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Publication:2045491
DOI10.3934/mbe.2019341zbMath1470.92074OpenAlexW2965881293WikidataQ91167868 ScholiaQ91167868MaRDI QIDQ2045491
Publication date: 13 August 2021
Published in: Mathematical Biosciences and Engineering (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.3934/mbe.2019341
global stabilityHIVantiretroviral therapyCTL immune responselatently infected cellscell-to-cell viral transmission
Epidemiology (92D30) Global stability of solutions to ordinary differential equations (34D23) Medical epidemiology (92C60) Pathology, pathophysiology (92C32)
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Cites Work
- Unnamed Item
- HIV-1 infection and low steady state viral loads
- Global stability and Hopf bifurcation of an HIV-1 infection model with saturation incidence and delayed CTL immune response
- Global threshold dynamics in a five-dimensional virus model with cell-mediated, humoral immune responses and distributed delays
- Dynamics of an HIV model with multiple infection stages and treatment with different drug classes
- Viral dynamics model with CTL immune response incorporating antiretroviral therapy
- The role of backward mutations on the within-host dynamics of HIV-1
- Global stability of a delayed HIV infection model with nonlinear incidence rate
- Global properties of a delayed HIV infection model with CTL immune response
- Global analysis of within host virus models with cell-to-cell viral transmission
- The effects of latent infection on the dynamics of HIV
- Modelling two different therapy strategies for drug T-20 on HIV-1 patients
- Global dynamics of a cell mediated immunity in viral infection models with distributed delays
- Stability and Hopf bifurcation of a HIV infection model with CTL-response delay
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Modeling HIV persistence, the latent reservoir, and viral blips
- Viral dynamics of an HIV model with latent infection incorporating antiretroviral therapy
- Mathematical analysis of a class of HIV infection models of \(\mathrm{CD4}^{+}\) T-cells with combined antiretroviral therapy
- Threshold dynamics of HIV-1 virus model with cell-to-cell transmission, cell-mediated immune responses and distributed delay
- Dynamics of HIV infection of CD4\(^ +\) T cells
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Global threshold dynamics in an HIV virus model with nonlinear infection rate and distributed invasion and production delays
- Persistence of low levels of plasma viremia and of the latent reservoir in patients under ART: a fractional-order approach
- Influence of raltegravir intensification on viral load and 2-LTR dynamics in HIV patients on suppressive antiretroviral therapy
- Emergence of HIV-1 drug resistance during antiretroviral treatment
- Optimal Control of an HIV Infection Model with Logistic Growth, CTL Immune Response and Infected Cells in Eclipse Phase