Latently infected cell activation: a way to reduce the size of the HIV reservoir?
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Publication:1758088
DOI10.1007/S11538-012-9729-XzbMath1251.92020OpenAlexW2084402697WikidataQ47311740 ScholiaQ47311740MaRDI QIDQ1758088
Stanca M. Ciupe, Joseph M. Volpe, Jonathan E. Forde
Publication date: 7 November 2012
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s11538-012-9729-x
Dynamical systems in biology (37N25) Medical applications (general) (92C50) Computational methods for problems pertaining to biology (92-08)
Related Items (6)
Periodic Solutions of an Infected-Age Structured HIV Model with the Latent Factor and Different Transmission Modes ⋮ Analysis of HIV models with two time delays ⋮ An HIV model with age-structured latently infected cells ⋮ Dynamics of a new HIV model with the activation status of infected cells ⋮ Stochastic population switch may explain the latent reservoir stability and intermittent viral blips in HIV patients on suppressive therapy ⋮ Virus Dynamics
Cites Work
- HIV-1 infection and low steady state viral loads
- Duration of an intermittent episode of viremia
- Can the viral reservoir of latently infected \(CD4^{+}\) T cells be eradicated with antiretroviral HIV drugs?
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Modeling HIV persistence, the latent reservoir, and viral blips
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