Impact of latently infected cells on strain archiving within HIV hosts
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Publication:692472
DOI10.1007/S11538-012-9742-0zbMath1256.92031OpenAlexW2076658665WikidataQ34331710 ScholiaQ34331710MaRDI QIDQ692472
Publication date: 6 December 2012
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s11538-012-9742-0
Probabilistic models, generic numerical methods in probability and statistics (65C20) Medical applications (general) (92C50)
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Cites Work
- HIV-1 infection and low steady state viral loads
- Opportunistic infection as a cause of transient viremia in chronically infected HIV patients under treatment with HAART
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Modeling HIV persistence, the latent reservoir, and viral blips
- Dynamics of HIV infection of CD4\(^ +\) T cells
- Emergence of HIV-1 drug resistance during antiretroviral treatment
- Drug concentration heterogeneity facilitates the evolution of drug resistance
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