Modeling secondary messenger pathways in neurovascular coupling

DOI10.1007/S11538-013-9813-XMaRDI QIDQ376431zbMATH OpenOpenAlexWikidataFDO

Authors James Hadfield, M. J. Plank, T. David

Publication date 5 November 2013

Published in Bulletin of Mathematical Biology (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1007/s11538-013-9813-x

zbMATH Keywords

arachidonic acidastrocyteautoregulationcalcium oscillationsglial cellsvasomotion

Mathematics Subject Classification ID

Computational methods for problems pertaining to biology (92-08) Neural biology (92C20) Cell biology (92C37) Biochemistry, molecular biology (92C40)

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Cites work

• Computational cell biology
• Models of neurovascular coupling via potassium and EET signalling
• On the origin and dynamics of the vasomotion of small arteries
• Origins of blood volume change due to glutamatergic synaptic activity at astrocytes abutting on arteriolar smooth muscle cells

Cited in

(6)

• The role of astrocytic calcium and TRPV4 channels in neurovascular coupling
• Modeling of ATP-mediated signal transduction and wave propagation in astrocytic cellular networks
• Neurovascular coupling and the influence of luminal agonists via the endothelium
• A bidirectional model for communication in the neurovascular unit
• scientific article; zbMATH DE number 7275073 (Why is no real title available?)
• A model of neurovascular coupling and its application to cortical spreading depolarization

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