B7-H1 and a mathematical model for cytotoxic T cell and tumor cell interaction
DOI10.1007/S11538-011-9665-1zbMATH Open1244.92030OpenAlexW2163579067WikidataQ51559918 ScholiaQ51559918MaRDI QIDQ417323FDOQ417323
Authors: Amanda Galante, Koji Tamada, Doron Levy 
Publication date: 14 May 2012
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s11538-011-9665-1
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Cites Work
- Modeling immunotherapy of the tumor -- immune interaction
- Cells behaving badly: A theoretical model for the fas/FasL system in tumour immunology
- The modelling of the immune competition by generalized kinetic (Boltzmann) models: Review and research perspectives
- A mathematical model of receptor-mediated apoptosis: Dying to know why FASL is a trimer
- Dynamics of granzyme B-induced apoptosis: Mathematical modeling
- Title not available (Why is that?)
Cited In (5)
- The union between structural and practical identifiability makes strength in reducing oncological model complexity: a case study
- A mathematical prognosis model for pancreatic cancer patients receiving immunotherapy
- Cells behaving badly: A theoretical model for the fas/FasL system in tumour immunology
- Modeling tumour heterogeneity of PD-L1 expression in tumour progression and adaptive therapy
- A mathematical model for pancreatic cancer growth and treatments
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