Modeling the role of TGF-\(\beta\) in regulation of the Th17 phenotype in the LPS-driven immune system

DOI10.1007/s11538-014-9946-6zbMath1297.92019WikidataQ51103663 ScholiaQ51103663MaRDI QIDQ458719

Seongwon Lee, Hyung-Ju Hwang, Yang-Jin Kim

Publication date: 8 October 2014

Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1007/s11538-014-9946-6

zbMATH Keywords

lipopolysaccharide; phenotypic switches; TGF-\(\beta\); Th17 phenotype

Mathematics Subject Classification ID

92C30: Physiology (general)

Related Items

Bifurcations and Multistability in a Model of Cytokine-Mediated Autoimmunity, Spatio-temporal models of lymphangiogenesis in wound healing, Hopf bifurcation in a model of TGF-\(\beta\) in regulation of the Th17 phenotype, Mathematical modeling and its analysis for instability of the immune system induced by chemotaxis

Uses Software

• Matlab

Cites Work

• Unnamed Item
• A hybrid model of tumor-stromal interactions in breast cancer
• Regulation of Th1/Th2 cells in asthma development: a mathematical model
• Interaction of tumor with its micro-environment: a mathematical model
• Th1/Th2 differentiation and cross-regulation
• A methodology for performing global uncertainty and sensitivity analysis in systems biology
• Combining cytokine signalling with T-bet and GATA-3 regulation in Th1 and Th2 differentiation: a model for cellular decision-making
• Mathematical modeling of allergy and specific immunotherapy: Th1-Th2-Treg interactions
• Multi-scale models of cell and tissue dynamics
• The role of the microenvironment in tumor invasion
• A HYBRID MODEL FOR TUMOR SPHEROID GROWTH IN VITRO I: THEORETICAL DEVELOPMENT AND EARLY RESULTS