Modeling the role of TGF- in regulation of the Th17 phenotype in the LPS-driven immune system
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Modeling the role of TGF-\(\beta\) in regulation of the Th17 phenotype in the LPS-driven immune system
Modeling the role of TGF-\(\beta\) in regulation of the Th17 phenotype in the LPS-driven immune system
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Cites work
- scientific article; zbMATH DE number 2115879 (Why is no real title available?)
- A HYBRID MODEL FOR TUMOR SPHEROID GROWTH IN VITRO I: THEORETICAL DEVELOPMENT AND EARLY RESULTS
- A hybrid model of tumor-stromal interactions in breast cancer
- A methodology for performing global uncertainty and sensitivity analysis in systems biology
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- Interaction of tumor with its micro-environment: a mathematical model
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Cited in
(10)- Bifurcations and multistability in a model of cytokine-mediated autoimmunity
- A dynamical model of TGF-β activation in asthmatic airways
- Regulation of Th1/Th2 cells in asthma development: a mathematical model
- Spatio-temporal models of lymphangiogenesis in wound healing
- Mathematical modeling and its analysis for instability of the immune system induced by chemotaxis
- Hopf bifurcation in a model of TGF-\(\beta\) in regulation of the Th17 phenotype
- Th1 or Th2: how an appropriate T helper response can be made
- Th1/Th2 differentiation and cross-regulation
- Combining cytokine signalling with T-bet and GATA-3 regulation in Th1 and Th2 differentiation: a model for cellular decision-making
- TH17 functional study in severe asthma using agent based model
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