On a multiscale model involving cell contractivity and its effects on tumor invasion
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Cites work
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- A finite difference scheme for a degenerated diffusion equation arising in microbial ecology
- A mathematical model for the effects of HER2 overexpression on cell proliferation in breast cancer
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- Multiscale models of taxis-driven patterning in bacterial populations
- Nonstandard finite difference schemes
- On a class of multiscale cancer cell migration models: well-posedness in less regular function spaces
- On some models for cancer cell migration through tissue networks
- On the asymptotic theory from microscopic to macroscopic growing tissue models: an overview with perspectives
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- \(M^5\) mesoscopic and macroscopic models for mesenchymal motion
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- Multiscale modeling of glioma invasion: from receptor binding to flux-limited macroscopic PDEs
- How far can chemotactic cross-diffusion enforce exceeding carrying capacities?
- Flux limitation mechanisms arising in multiscale modelling of cancer invasion
- On a structured multiscale model for acid-mediated tumor invasion: the effects of adhesion and proliferation
- Mathematical research for models which is related to chemotaxis system
- Finite-time blow-up in low-dimensional Keller-Segel systems with logistic-type superlinear degradation
- Critical mass for infinite-time aggregation in a chemotaxis model with indirect signal production
- Mathematical modeling and numerical simulation of a multiscale cancer invasion of host tissue
- Boundedness of solutions to a quasilinear higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- Multi-level mathematical models for cell migration in confined environments
- Global existence of classical solutions to a chemotaxis-haptotaxis model
- On a coupled SDE-PDE system modeling acid-mediated tumor invasion
- A multiscale modeling approach to glioma invasion with therapy
- Modeling multiple taxis: tumor invasion with phenotypic heterogeneity, haptotaxis, and unilateral interspecies repellence
- Glioma follow white matter tracts: a multiscale DTI-based model
- Tumor cells proliferation and migration under the influence of their microenvironment
- Emergence of large population densities despite logistic growth restrictions in fully parabolic chemotaxis systems
- How nucleus mechanics and ECM microstructure influence the invasion of single cells and multicellular aggregates
- Global asymptotic stability of constant equilibria in a fully parabolic chemotaxis system with strong logistic dampening
- Mini-workshop: Mathematical models for cancer cell migration. Abstracts from the mini-workshop held April 13--19, 2014.
- Global existence for a degenerate haptotaxis model of cancer invasion
- DRBEM-FDM solution of a chemotaxis-haptotaxis model for cancer invasion
- Glioma invasion and its interplay with nervous tissue and therapy: a multiscale model
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