Stability and bifurcation analysis of an HIV-1 infection model with a general incidence and CTL immune response
From MaRDI portal
Publication:5862011
DOI10.1080/17513758.2021.1950224zbMATH Open1484.92125OpenAlexW3178761750MaRDI QIDQ5862011FDOQ5862011
Yuming Chen, Yuhuai Zhang, Xinsheng Ma
Publication date: 4 March 2022
Published in: Journal of Biological Dynamics (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1080/17513758.2021.1950224
Recommendations
- Global stability for an HIV-1 infection model with Beddington-DeAngelis incidence rate and CTL immune response
- Global stability and Hopf bifurcation of an HIV-1 infection model with saturation incidence and delayed CTL immune response
- Stability of HIV-1 infection with saturated virus-target and infected-target incidences and CTL immune response
- Stability and Hopf bifurcation in a delayed HIV infection model with general incidence rate and immune impairment
- Global stability of an HIV infection model with saturated CTL immune response and intracellular delay
Epidemiology (92D30) Bifurcation theory for ordinary differential equations (34C23) Global stability of solutions to ordinary differential equations (34D23)
Cites Work
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Dynamical models of tuberculosis and their applications
- Analysis of a CD4\(^+\) T cell viral infection model with a class of saturated infection rate
- Stability by Liapunov's direct method. With applications
- Global stability and periodic solution of the viral dynamics
- HIV drug resistance: insights from mathematical modelling
- Modeling within-host HIV-1 dynamics and the evolution of drug resistance: trade-offs between viral enzyme function and drug susceptibility
- Mathematical analysis of the transmission dynamics of HIV syphilis co-infection in the presence of treatment for syphilis
- Modeling cell-to-cell spread of HIV-1 with logistic target cell growth
- A delayed HIV-1 model with virus waning term
- Global stability for an HIV-1 infection model with Beddington-DeAngelis incidence rate and CTL immune response
- The existence of codimension-two bifurcation in a discrete SIS epidemic model with standard incidence
- Global stability for an HIV-1 infection model including an eclipse stage of infected cells
- Global stability of an infection-age structured HIV-1 model linking within-host and between-host dynamics
- Title not available (Why is that?)
- The role of CD4 T cells in immune system activation and viral reproduction in a simple model for HIV infection
- Optimal treatment in a multi-strain within-host model of HIV with age structure
- An age-structured model for the coupled dynamics of HIV and HSV-2
- Title not available (Why is that?)
- Title not available (Why is that?)
- Stability of a general HIV-1 reaction-diffusion model with multiple delays and immune response
- Drug therapy model with time delays for HIV infection with virus-to-cell and cell-to-cell transmissions
- EFFECTS OF ECLIPSE PHASE AND DELAY ON THE DYNAMICS OF HIV INFECTION
- Dynamical properties and thresholds of an HIV model with super-infection
- Title not available (Why is that?)
Cited In (12)
- Stability of HIV-1 infection with saturated virus-target and infected-target incidences and CTL immune response
- Bifurcation Analysis for Two-Species Commensalism (Amensalism) Systems with Distributed Delays
- Analysis of equilibria and connecting orbits in a nonlinear viral infection model
- Dynamic analysis of HIV model with a general incidence, CTLs immune response and intracellular delays
- Title not available (Why is that?)
- Stability of a general CTL-mediated immunity HIV infection model with silent infected cell-to-cell spread
- Bifurcation analysis of HIV infection model with antibody and cytotoxic T‐lymphocyte immune responses and Beddington–DeAngelis functional response
- Stability analysis of general humoral immunity HIV dynamics models with discrete delays and HAART
- Stability analysis of a general HIV dynamics model with multi-stages of infected cells and two routes of infection
- Backward bifurcation of an HTLV-I model with immune response
- Title not available (Why is that?)
- Stability and zero-Hopf bifurcation analysis of a tumour and T-helper cells interaction model in the case of HIV infection
This page was built for publication: Stability and bifurcation analysis of an HIV-1 infection model with a general incidence and CTL immune response
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q5862011)
