Stability and bifurcation analysis of an HIV-1 infection model with a general incidence and CTL immune response
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Publication:5862011
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Cites work
- scientific article; zbMATH DE number 3464555 (Why is no real title available?)
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- Global stability for an HIV-1 infection model including an eclipse stage of infected cells
- Global stability for an HIV-1 infection model with Beddington-DeAngelis incidence rate and CTL immune response
- Global stability of an infection-age structured HIV-1 model linking within-host and between-host dynamics
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Cited in
(16)- scientific article; zbMATH DE number 7458923 (Why is no real title available?)
- Stability analysis of a general HIV dynamics model with multi-stages of infected cells and two routes of infection
- scientific article; zbMATH DE number 7404456 (Why is no real title available?)
- An in-host HIV-1 infection model incorporating quiescent and activated \(\mathrm{CD}4^+\) T cells as well as CTL response
- Stability of a general CTL-mediated immunity HIV infection model with silent infected cell-to-cell spread
- Backward bifurcation of an HTLV-I model with immune response
- Bifurcation analysis for two-species commensalism (amensalism) systems with distributed delays
- Transcritical bifurcation of an immunosuppressive infection model
- Stability of HIV-1 infection with saturated virus-target and infected-target incidences and CTL immune response
- Bifurcation analysis of the HIV-1 within host model
- Detecting and resetting tipping points to create more HIV post-treatment controllers with bifurcation and sensitivity analysis
- Bifurcation analysis of HIV infection model with antibody and cytotoxic T‐lymphocyte immune responses and Beddington–DeAngelis functional response
- Dynamic analysis of HIV model with a general incidence, CTLs immune response and intracellular delays
- Stability analysis of general humoral immunity HIV dynamics models with discrete delays and HAART
- Analysis of equilibria and connecting orbits in a nonlinear viral infection model
- Stability and zero-Hopf bifurcation analysis of a tumour and T-helper cells interaction model in the case of HIV infection
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