Global stability for an HIV-1 infection model including an eclipse stage of infected cells

From MaRDI portal
Publication:641632


DOI10.1016/j.jmaa.2011.07.006zbMath1223.92024WikidataQ91899968 ScholiaQ91899968MaRDI QIDQ641632

Bruno Buonomo, Cruz Vargas-De-León

Publication date: 24 October 2011

Published in: Journal of Mathematical Analysis and Applications (Search for Journal in Brave)

Full work available at URL: http://europepmc.org/articles/pmc7127580


65C20: Probabilistic models, generic numerical methods in probability and statistics

92C50: Medical applications (general)

34C60: Qualitative investigation and simulation of ordinary differential equation models

34D23: Global stability of solutions to ordinary differential equations


Related Items

Unnamed Item, Stability of a CD4+ T cell viral infection model with diffusion, Stability of general virus dynamics models with both cellular and viral infections, Accounting for multi-delay effects in an HIV-1 infection model with saturated infection rate, recovery and proliferation of host cells, Stability and Hopf bifurcation of HIV-1 model with Holling II infection rate and immune delay, Global Dynamics of a Vector-Borne Disease Model with Two Transmission Routes, Modeling and stability analysis of HIV/HTLV-I co-infection, Global analysis of virus dynamics model with logistic mitosis, cure rate and delay in virus production, Stability analysis of general humoral immunity HIV dynamics models with discrete delays and HAART, Optimal Control of an HIV Infection Model with Logistic Growth, CTL Immune Response and Infected Cells in Eclipse Phase, Global stability of an SEIQV epidemic model with general incidence rate, Stability of HIV-1 infection with saturated virus-target and infected-target incidences and CTL immune response, Stability and bifurcation analysis of an HIV-1 infection model with a general incidence and CTL immune response, Analysis and optimal control of an HIV model with logistic growth and infected cells in eclipse phase, Stability and Hopf bifurcation in a delayed HIV infection model with general incidence rate and immune impairment, Immune response in virus model structured by cell infection-age, Role of \(CD4^{+}\) T-cell proliferation in HIV infection under antiretroviral therapy, Backward bifurcation and global stability in an epidemic model with treatment and vaccination, Modeling of pseudo-rational exemption to vaccination for SEIR diseases, Global dynamics for viral infection model with Beddington-Deangelis functional response and an eclipse stage of infected cells, Global stability of infectious disease models with contact rate as a function of prevalence index, Global dynamics of a SEIRS epidemic model with saturated disease transmission rate and vaccination control, Global stability of humoral immunity virus dynamics models with nonlinear infection rate and removal, Global dynamics for an HIV infection model with Crowley-Martin functional response and two distributed delays, Stability and bifurcation in plant-pathogens interactions, Global properties of general viral infection models with humoral immune response, Global analysis of a mathematical model for hepatitis C considering the host immune system, Effect of cellular reservoirs and delays on the global dynamics of HIV, Stability of delayed HIV dynamics models with two latent reservoirs and immune impairment, Numerical analysis for a fractional differential time-delay model of HIV infection of CD4\(^+\) T-cell proliferation under antiretroviral therapy, Threshold dynamics of an HIV-1 virus model with both virus-to-cell and cell-to-cell transmissions, intracellular delay, and humoral immunity, A mathematical analysis of pine wilt disease with variable population size and optimal control strategies, Stability of an adaptive immunity delayed HIV infection model with active and silent cell-to-cell spread, Modeling the role of acquired immune response and antiretroviral therapy in the dynamics of HIV infection, Impacts of media awareness on a stage structured epidemic model, Stability and dynamic of HIV-1 mathematical model with logistic target cell growth, treatment rate, cure rate and cell-to-cell spread, An epidemiological approach to insurgent population modeling with the Atangana-Baleanu fractional derivative, Lyapunov functions for fractional-order systems in biology: methods and applications, Stability of general pathogen dynamic models with two types of infectious transmission with immune impairment, Mathematical modeling of HIV/HTLV co-infection with CTL-mediated immunity, Complex dynamics of an SEIR epidemic model with saturated incidence rate and treatment, Asymptotic and stability dynamics of an HIV-1-cytotoxic T lymphocytes (CTL) chemotaxis model, Stability and bifurcation analysis of an epidemic model with the effect of media, Global stability and persistence of HIV models with switching parameters and pulse control, Mathematical analysis of the impact of transmission-blocking drugs on the population dynamics of malaria, Stability analysis of a model for HBV infection with cure of infected cells and intracellular delay, Dynamics analysis and simulation of a modified HIV infection model with a saturated infection rate, Global properties of a general latent pathogen dynamics model with delayed pathogenic and cellular infections, Resonance of periodic combination antiviral therapy and intracellular delays in virus model, Global stability analysis of HIV-1 infection model with three time delays, Dynamics analysis of an HIV infection model including infected cells in an eclipse stage, Analysis of a CD4\(^+\) T cell viral infection model with a class of saturated infection rate, Existence theory and numerical simulation of HIV-I cure model with new fractional derivative possessing a non-singular kernel, Stability of a general delay‐distributed virus dynamics model with multi‐staged infected progression and immune response, GLOBAL STABILITY OF A GENERAL VIRUS DYNAMICS MODEL WITH MULTI-STAGED INFECTED PROGRESSION AND HUMORAL IMMUNITY, Hopf bifurcation of an age-structured HIV infection model with logistic target-cell growth, Global properties of nonlinear humoral immunity viral infection models, Global dynamics of delay‐distributed HIV infection models with differential drug efficacy in cocirculating target cells



Cites Work