Role of CD4^+ T-cell proliferation in HIV infection under antiretroviral therapy
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Publication:442473
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Cites work
- scientific article; zbMATH DE number 17855 (Why is no real title available?)
- scientific article; zbMATH DE number 3568468 (Why is no real title available?)
- scientific article; zbMATH DE number 2038872 (Why is no real title available?)
- A Geometric Approach to Global-Stability Problems
- A simple SIS epidemic model with a backward bifurcation
- A theory of polytopes
- Backward bifurcation in a model for HTLV-I infection of CD4\(^{+}\) T cells
- Backwards bifurcations and catastrophe in simple models of fatal diseases
- Complex patterns of viral load decay under antiretroviral therapy: influence of pharmacokinetics and intracellular delay
- Compound matrices and ordinary differential equations
- Drug concentration heterogeneity facilitates the evolution of drug resistance
- Fighting a virus with a virus: A dynamic model for HIV-1 therapy
- Global Results for an Epidemic Model with Vaccination that Exhibits Backward Bifurcation
- Global dynamics of a SEIR model with varying total population size
- Global dynamics of a mathematical model for HTLV-I infection of \(CD4^{+}T\) cells with delayed CTL response
- Global properties of basic virus dynamics models
- Global stability for an HIV-1 infection model including an eclipse stage of infected cells
- Global stability of an HIV pathogenesis model with cure rate
- Global stability of an HIV-1 infection model with saturation infection and intracellular delay
- Global stability of an HIV-1 model with distributed intracellular delays and a combination therapy
- Infection dynamics in HIV-specific CD4 T cells: does a CD4 T cell boost benefit the host or the virus?
- Influence of backward bifurcation in a model of hepatitis B and C viruses
- Joint effects of mitosis and intracellular delay on viral dynamics: two-parameter bifurcation analysis
- Logarithmic norms and projections applied to linear differential systems
- Mathematical Analysis of HIV-1 Dynamics in Vivo
- Mathematical analysis of the global dynamics of a model for HIV infection of CD4\(^{+}\) T cells
- Monte Carlo estimates of natural variation in HIV infection
- On Bendixson's criterion
- Permanence and extinction of a nonautonomous HIV/AIDS epidemic model with distributed time delay
- Persistence in dynamical systems
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Restricted occupancy models for neutralization of HIV virions and populations
- Role of incidence function in vaccine-induced backward bifurcation in some HIV models
- Uniform persistence and flows near a closed positively invariant set
- Using mathematics to understand HIV immune dynamics.
- Virus Dynamics: A Global Analysis
Cited in
(20)- A class of virus dynamic model with inhibitory effect on the growth of uninfected T cells caused by infected T cells and its stability analysis
- Numerical analysis for a fractional differential time-delay model of HIV infection of CD4\(^+\) T-cell proliferation under antiretroviral therapy
- Kinetic model of HIV infection including hematopoietic progenitor cells
- Insight of T cell proliferation in the estimation of expected time to extinction of the disease HIV/AIDS
- Bifurcations of a mathematical model for HIV dynamics
- An HIV infection model based on a vectored immunoprophylaxis experiment
- Global dynamics of an age-structured virus model with saturation effects
- Hopf bifurcation and stability of periodic solutions for delay differential model of HIV infection of CD4 T-cells
- Backward bifurcation in a mathematical model for HIV infection \textit{in vivo} with anti-retroviral treatment
- Backward bifurcation and global stability in an epidemic model with treatment and vaccination
- Complete classification of global dynamics of a virus model with immune responses
- Global stability and backward bifurcation of a general viral infection model with virus-driven proliferation of target cells
- Modeling virus-stimulated proliferation of CD \(4^+\) T-cell, cell-to-cell transmission and viral loss in HIV infection dynamics
- How self-proliferation of CD4\(^{+}\)T cells affect the HIV dynamics in an in-host target-cell limited HIV model with saturation infection rate: a quasi-steady-state approximation analysis
- Infection dynamics in HIV-specific CD4 T cells: does a CD4 T cell boost benefit the host or the virus?
- HIV infection and CD\(4^+\) T cell dynamics
- Global dynamics of a delay virus model with recruitment and saturation effects of immune responses
- Stability and bifurcation in plant-pathogens interactions
- Modeling the effect of activation of CD\(4^+\) T cells on HIV dynamics
- Dynamics analysis of a delayed virus model with two different transmission methods and treatments
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