Role of \(CD4^{+}\) T-cell proliferation in HIV infection under antiretroviral therapy
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Publication:442473
DOI10.1016/j.jmaa.2012.05.027zbMath1275.92033OpenAlexW2047656200MaRDI QIDQ442473
Publication date: 1 August 2012
Published in: Journal of Mathematical Analysis and Applications (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jmaa.2012.05.027
Dynamical systems in biology (37N25) Bifurcation theory for ordinary differential equations (34C23) Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60) Medical epidemiology (92C60)
Related Items (12)
An HIV infection model based on a vectored immunoprophylaxis experiment ⋮ Global dynamics of a delay virus model with recruitment and saturation effects of immune responses ⋮ Dynamics analysis of a delayed virus model with two different transmission methods and treatments ⋮ Stability and bifurcation in plant-pathogens interactions ⋮ Bifurcations of a mathematical model for HIV dynamics ⋮ Global dynamics of an age-structured virus model with saturation effects ⋮ Numerical analysis for a fractional differential time-delay model of HIV infection of CD4\(^+\) T-cell proliferation under antiretroviral therapy ⋮ Hopf bifurcation and stability of periodic solutions for delay differential model of HIV infection of CD4 T-cells ⋮ Backward bifurcation and global stability in an epidemic model with treatment and vaccination ⋮ Complete classification of global dynamics of a virus model with immune responses ⋮ Global stability and backward bifurcation of a general viral infection model with virus-driven proliferation of target cells ⋮ A class of virus dynamic model with inhibitory effect on the growth of uninfected T cells caused by infected T cells and its stability analysis
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