Role of CD4^+ T-cell proliferation in HIV infection under antiretroviral therapy
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Publication:442473
DOI10.1016/J.JMAA.2012.05.027zbMATH Open1275.92033OpenAlexW2047656200MaRDI QIDQ442473FDOQ442473
Authors: Hongying Shu, Lin Wang 
Publication date: 1 August 2012
Published in: Journal of Mathematical Analysis and Applications (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jmaa.2012.05.027
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Medical applications (general) (92C50) Medical epidemiology (92C60) Qualitative investigation and simulation of ordinary differential equation models (34C60) Dynamical systems in biology (37N25) Bifurcation theory for ordinary differential equations (34C23)
Cites Work
- Title not available (Why is that?)
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Complex patterns of viral load decay under antiretroviral therapy: influence of pharmacokinetics and intracellular delay
- Global properties of basic virus dynamics models
- Title not available (Why is that?)
- Mathematical Analysis of HIV-1 Dynamics in Vivo
- Virus Dynamics: A Global Analysis
- Global stability of an HIV-1 infection model with saturation infection and intracellular delay
- A simple SIS epidemic model with a backward bifurcation
- Global dynamics of a SEIR model with varying total population size
- Compound matrices and ordinary differential equations
- Backward bifurcation in a model for HTLV-I infection of CD4\(^{+}\) T cells
- Global Results for an Epidemic Model with Vaccination that Exhibits Backward Bifurcation
- A Geometric Approach to Global-Stability Problems
- Logarithmic norms and projections applied to linear differential systems
- On Bendixson's criterion
- Uniform persistence and flows near a closed positively invariant set
- Using mathematics to understand HIV immune dynamics.
- Global dynamics of a mathematical model for HTLV-I infection of \(CD4^{+}T\) cells with delayed CTL response
- Title not available (Why is that?)
- Persistence in dynamical systems
- Global stability of an HIV pathogenesis model with cure rate
- Monte Carlo estimates of natural variation in HIV infection
- Backwards bifurcations and catastrophe in simple models of fatal diseases
- Mathematical analysis of the global dynamics of a model for HIV infection of CD4\(^{+}\) T cells
- Joint effects of mitosis and intracellular delay on viral dynamics: two-parameter bifurcation analysis
- Influence of backward bifurcation in a model of hepatitis B and C viruses
- Role of incidence function in vaccine-induced backward bifurcation in some HIV models
- Drug concentration heterogeneity facilitates the evolution of drug resistance
- Global stability for an HIV-1 infection model including an eclipse stage of infected cells
- Permanence and extinction of a nonautonomous HIV/AIDS epidemic model with distributed time delay
- Global stability of an HIV-1 model with distributed intracellular delays and a combination therapy
- Fighting a virus with a virus: A dynamic model for HIV-1 therapy
- Restricted occupancy models for neutralization of HIV virions and populations
- Infection dynamics in HIV-specific CD4 T cells: does a CD4 T cell boost benefit the host or the virus?
- A theory of polytopes
Cited In (20)
- A class of virus dynamic model with inhibitory effect on the growth of uninfected T cells caused by infected T cells and its stability analysis
- Numerical analysis for a fractional differential time-delay model of HIV infection of CD4\(^+\) T-cell proliferation under antiretroviral therapy
- Kinetic model of HIV infection including hematopoietic progenitor cells
- Insight of T cell proliferation in the estimation of expected time to extinction of the disease HIV/AIDS
- Bifurcations of a mathematical model for HIV dynamics
- An HIV infection model based on a vectored immunoprophylaxis experiment
- Global dynamics of an age-structured virus model with saturation effects
- Hopf bifurcation and stability of periodic solutions for delay differential model of HIV infection of CD4 T-cells
- Backward bifurcation in a mathematical model for HIV infection \textit{in vivo} with anti-retroviral treatment
- Backward bifurcation and global stability in an epidemic model with treatment and vaccination
- Complete classification of global dynamics of a virus model with immune responses
- Global stability and backward bifurcation of a general viral infection model with virus-driven proliferation of target cells
- Modeling virus-stimulated proliferation of CD \(4^+\) T-cell, cell-to-cell transmission and viral loss in HIV infection dynamics
- How self-proliferation of CD4\(^{+}\)T cells affect the HIV dynamics in an in-host target-cell limited HIV model with saturation infection rate: a quasi-steady-state approximation analysis
- Infection dynamics in HIV-specific CD4 T cells: does a CD4 T cell boost benefit the host or the virus?
- HIV infection and CD\(4^+\) T cell dynamics
- Global dynamics of a delay virus model with recruitment and saturation effects of immune responses
- Stability and bifurcation in plant-pathogens interactions
- Modeling the effect of activation of CD\(4^+\) T cells on HIV dynamics
- Dynamics analysis of a delayed virus model with two different transmission methods and treatments
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