Global stability and backward bifurcation of a general viral infection model with virus-driven proliferation of target cells
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Publication:478668
DOI10.3934/DCDSB.2014.19.1749zbMATH Open1327.92066OpenAlexW2317078136MaRDI QIDQ478668FDOQ478668
Authors: Hongying Shu, Lin Wang 
Publication date: 4 December 2014
Published in: Discrete and Continuous Dynamical Systems. Series B (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.3934/dcdsb.2014.19.1749
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Cites Work
- Title not available (Why is that?)
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Introduction to functional differential equations
- Complex patterns of viral load decay under antiretroviral therapy: influence of pharmacokinetics and intracellular delay
- Stability analysis of pathogen-immune interaction dynamics
- Global properties of basic virus dynamics models
- Title not available (Why is that?)
- Mathematical Analysis of HIV-1 Dynamics in Vivo
- Role of \(CD4^{+}\) T-cell proliferation in HIV infection under antiretroviral therapy
- Virus Dynamics: A Global Analysis
- Dynamics of a HIV-1 infection model with cell-mediated immune response and intracellular delay
- Global dynamics of a SEIR model with varying total population size
- Compound matrices and ordinary differential equations
- Dynamical systems: Stability theory and applications
- A Geometric Approach to Global-Stability Problems
- Logarithmic norms and projections applied to linear differential systems
- On Bendixson's criterion
- Uniform persistence and flows near a closed positively invariant set
- Using mathematics to understand HIV immune dynamics.
- Title not available (Why is that?)
- Impact of intracellular delays and target-cell dynamics on in vivo viral infections
- Title not available (Why is that?)
- Persistence in dynamical systems
- Global stability of a five-dimensional model with immune responses and delay
- Monte Carlo estimates of natural variation in HIV infection
- Mathematical analysis of the global dynamics of a model for HIV infection of CD4\(^{+}\) T cells
- Joint effects of mitosis and intracellular delay on viral dynamics: two-parameter bifurcation analysis
- Influence of backward bifurcation in a model of hepatitis B and C viruses
- Drug concentration heterogeneity facilitates the evolution of drug resistance
- HIV infection and CD\(4^+\) T cell dynamics
- Fighting a virus with a virus: A dynamic model for HIV-1 therapy
Cited In (4)
- Bifurcations of a mathematical model for HIV dynamics
- Global stability of a diffusive and delayed virus dynamics model with Beddington-DeAngelis incidence function and CTL immune response
- Modelling HIV dynamics with cell‐to‐cell transmission and CTL response
- Global analysis of a multi-group viral infection model with age structure
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