The growth rate of senile plaques is determined by the competition between the rate of deposition of free A aggregates into plaques and the autocatalytic production of free A aggregates
From MaRDI portal
Publication:6594289
Recommendations
- Numerical modeling of senile plaque development under conditions of limited diffusivity of amyloid-\(\beta\) monomers
- Simulating the effect of formation of amyloid plaques on aggregation of tau protein
- Modeling effect of a \(\gamma \)-secretase inhibitor on amyloid-\(\beta \) dynamics reveals significant role of an amyloid clearance mechanism
- A mathematical model demonstrating the role of interstitial fluid flow on the clearance and accumulation of amyloid \(\beta\) in the brain
Cites work
- scientific article; zbMATH DE number 3565945 (Why is no real title available?)
- A class of exact solutions for biomacromolecule diffusion-reaction in live cells
- An analytical solution simulating growth of Lewy bodies
- How the formation of amyloid plaques and neurofibrillary tangles may be related: a mathematical modelling study
- Numerical modeling of senile plaque development under conditions of limited diffusivity of amyloid-\(\beta\) monomers
- Simulating the effect of formation of amyloid plaques on aggregation of tau protein
Cited in
(2)
This page was built for publication: The growth rate of senile plaques is determined by the competition between the rate of deposition of free A\(\beta\) aggregates into plaques and the autocatalytic production of free A\(\beta\) aggregates
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q6594289)