A simple cellular automaton model for influenza A viral infections
From MaRDI portal
Publication:776479
DOI10.1016/j.jtbi.2004.08.001zbMath1442.92153arXivq-bio/0402012OpenAlexW1965004987WikidataQ47866416 ScholiaQ47866416MaRDI QIDQ776479
John Samuel, Jack A. Tuszyński, Catherine A. A. Beauchemin
Publication date: 9 July 2020
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://arxiv.org/abs/q-bio/0402012
Related Items
Stationary states and spatial patterning in an \(S I S\) epidemiology model with implicit mobility, Within-host demographic fluctuations and correlations in early retroviral infection, A single-cell mathematical model of SARS-CoV-2 induced pyroptosis and the effects of anti-inflammatory intervention, A hybrid stochastic-deterministic computational model accurately describes spatial dynamics and virus diffusion in HIV-1 growth competition assay, Predicting pathogen-specific CD8 T cell immune responses from a modeling approach, Towards multiscale modeling of influenza infection, Dynamics of influenza virus and human host interactions during infection and replication cycle, Modeling epidemics using cellular automata, Rival approaches to mathematical modelling in immunology, A Review of Multi‐Compartment Infectious Disease Models, Ex vivo experiments shed light on the innate immune response from influenza virus, Probing the effects of the well-mixed assumption on viral infection dynamics, Simple scaling laws for influenza A rise time, duration, and severity, On the derivation of approximations to cellular automata models and the assumption of independence, A multiscale multicellular spatiotemporal model of local influenza infection and immune response, A self-organizing criticality mathematical model for contamination and epidemic spreading, Equation-free multiscale computational analysis of individual-based epidemic dynamics on networks, Real Linear Automata with a Continuum of Periodic Solutions for Every Period, The rate of viral transfer between upper and lower respiratory tracts determines RSV illness duration, Spreading processes in post-epidemic environments
