Evolutionary dynamics of tumor progression with random fitness values
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Publication:1630905
DOI10.1016/j.tpb.2010.05.001zbMath1403.92172arXiv1003.1927OpenAlexW3103521600WikidataQ42451818 ScholiaQ42451818MaRDI QIDQ1630905
Jasmine Foo, Kevin Leder, Franziska Michor, Richard T. Durrett, J. P. Mayberry
Publication date: 5 December 2018
Published in: Theoretical Population Biology (Search for Journal in Brave)
Full work available at URL: https://arxiv.org/abs/1003.1927
Problems related to evolution (92D15) Applications of branching processes (60J85) Medical applications (general) (92C50) Genetics and epigenetics (92D10)
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Cites Work
- Traveling waves of selective sweeps
- Population genetics of tumor suppressor genes
- Analysis of tracer particle migration in inhomogeneous turbulence
- A waiting time problem arising from the study of multi-stage carcinogenesis
- Waiting for \(m\) mutations
- Optimal control for a stochastic model of cancer chemotherapy
- Evolution of resistance and progression to disease during clonal expansion of cancer
- A simple stochastic gene substitution model
- Genetic instability and clonal expansion
- Dynamics of metastasis suppressor gene inactivation
- Stochastic dynamics of metastasis formation
- Estimation of Parameters and Larger Quantiles Based on the k Largest Observations
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