Multispecies model of cell lineages and feedback control in solid tumors
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Publication:1784366
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Cites work
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- The tumor growth paradox and immune system-mediated selection for cancer stem cells
- Three-dimensional multispecies nonlinear tumor growth. I: Model and numerical method
- Three-dimensional multispecies nonlinear tumor growth. II: Tumor invasion and angiogenesis
Cited in
(19)- A multiphase Cahn-Hilliard-Darcy model for tumour growth with necrosis
- A multicompartment mathematical model of cancer stem cell-driven tumor growth dynamics
- Stability of control networks in autonomous homeostatic regulation of stem cell lineages
- The tumor growth paradox and immune system-mediated selection for cancer stem cells
- Mathematical modeling of tumor and cancer stem cells treated with CAR-T therapy and inhibition of TGF-\(\beta\)
- Activation of the HGF/c-met axis in the tumor microenvironment: a multispecies model
- Feedback regulation in a cancer stem cell model can cause an Allee effect
- Mathematical modeling of the role of \textit{survivin} on dedifferentiation and radioresistance in cancer
- Three-dimensional spatiotemporal modeling of colon cancer organoids reveals that multimodal control of stem cell self-renewal is a critical determinant of size and shape in early stages of tumor growth
- Determining the control networks regulating stem cell lineages in colonic crypts
- Continuum approximation of invasion probabilities
- Tumours with cancer stem cells: a PDE model
- Modelling of cancer stem cell driven solid tumors
- Effect of feedback regulation on stem cell fractions in tissues and tumors: understanding chemoresistance in cancer
- Mathematical characterization of heterogeneity in a cancer stem cell driven tumor growth model with nonlinear self-renewal
- Interlocking triads of growth control in tumors
- A multi-phenotypic cancer model with cell plasticity
- Modeling tumorspheres reveals cancer stem cell niche building and plasticity
- On the onset of multicellular invasive behavior in hierarchical lineage: the role of inhibitory feedback and local fluctuations
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