A microscale mathematical model for metabolic symbiosis: investigating the effects of metabolic inhibition on ATP turnover in tumors
DOI10.1016/J.JTBI.2014.11.016zbMATH Open1412.92102OpenAlexW2048652043WikidataQ51006461 ScholiaQ51006461MaRDI QIDQ2415751FDOQ2415751
Mohammad Kohandel, Colin Phipps, Hamid Molavian
Publication date: 23 May 2019
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2014.11.016
:tumor hypoxiacancer cell metabolismglycolytic inhibitionlactate dehydrogenase inhibitorwarburg effect
Biochemistry, molecular biology (92C40) Kinetics in biochemical problems (pharmacokinetics, enzyme kinetics, etc.) (92C45)
Cites Work
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- A mathematical model for the glucose-lactate metabolism of in vitro cancer cells
- Necrotic core in EMT6/Ro tumour spheroids: is it caused by an ATP deficit?
- Incorporating energy metabolism into a growth model of multicellular tumor spheroids
- Cell resensitization after delivery of a cycle-specific anticancer drug and effect of dose splitting: learning from tumour cords
- ATP production and necrosis formation in a tumour spheroid model
Cited In (5)
- A mathematical model for the glucose-lactate metabolism of in vitro cancer cells
- Glucose-lactate metabolic cooperation in cancer: insights from a spatial mathematical model and implications for targeted therapy
- A theoretical explanation of the laws of Warburg and Sigmond
- A reduced model of cell metabolism to revisit the glycolysis-OXPHOS relationship in the deregulated tumor microenvironment
- Incorporating energy metabolism into a growth model of multicellular tumor spheroids
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