Modeling inoculum dose dependent patterns of acute virus infections
From MaRDI portal
Publication:2632610
DOI10.1016/j.jtbi.2014.01.008zbMath1412.92290OpenAlexW2065820141WikidataQ39269196 ScholiaQ39269196MaRDI QIDQ2632610
Publication date: 15 May 2019
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2014.01.008
Related Items
A mathematical framework for predicting lifestyles of viral pathogens ⋮ A target-cell limited model can reproduce influenza infection dynamics in hosts with differing immune responses ⋮ What controls the acute viral infection following yellow fever vaccination? ⋮ Mathematical modelling of SARS-CoV-2 infection of human and animal host cells reveals differences in the infection rates and delays in viral particle production by infected cells ⋮ Modelling immune memory development ⋮ Varying inoculum dose to assess the roles of the immune response and target cell depletion by the pathogen in control of acute viral infections ⋮ The rate of viral transfer between upper and lower respiratory tracts determines RSV illness duration ⋮ The role of a programmatic immune response on the evolution of pathogen traits
Uses Software
Cites Work
- Unnamed Item
- An accurate two-phase approximate solution to an acute viral infection model
- An in-host model of acute infection: measles as a case study
- Killer cell dynamics. Mathematical and computational approaches to immunology.
- Modeling immune responses with handling time
- Optimal strategies in immunology. I: B-cell differentiation and proliferation
- Exploring the role of the immune response in preventing antibiotic resistance
- Modeling amantadine treatment of influenza A virus in vitro
- Extending the quasi-steady state approximation by changing variables
- Models of CD8+ responses: 1. what is the antigen-independent proliferation program
- A dynamical model of human immune response to influenza A virus infection
- Mathematical model of a three-stage innate immune response to a pneumococcal lung infection
- The Quasi-Steady-State Assumption: A Case Study in Perturbation