Mathematical model of mycobacterium-host interaction describes physiology of persistence

DOI10.1016/J.JTBI.2015.03.031zbMATH Open1347.92044OpenAlexW2094204987WikidataQ41341690 ScholiaQ41341690MaRDI QIDQ327128FDOQ327128

Authors: Gabriele Pedruzzi, Kanury V. S. Rao, S. Chatterjee 

Publication date: 19 October 2016

Published in: Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1016/j.jtbi.2015.03.031

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zbMATH Keywords

differential equationsnitric oxidehost-pathogen interactionlipidsiron

Mathematics Subject Classification ID

Medical applications (general) (92C50) Medical epidemiology (92C60) Epidemiology (92D30)

Cites Work

• A Comparison of Three Methods for Selecting Values of Input Variables in the Analysis of Output from a Computer Code
• The mathematics of infectious diseases
• Criterion of Hopf bifurcations without using eigenvalues
• A methodology for performing global uncertainty and sensitivity analysis in systems biology
• Title not available (Why is that?)
• The timing of TNF and IFN-\(\gamma\) signaling affects macrophage activation strategies during \textit{Mycobacterium tuberculosis} infection
• A model for tuberculosis with exogenous reinfection
• A mathematical representation of the development of \textit{Mycobacterium tuberculosis} active, latent and dormant stages

Cited In (13)

• A mathematical model to study low-dose metronomic scheduling for chemotherapy
• Deciphering the virulence of \textit{Mycobacterium avium} subsp. \textit{paratuberculosis} isolates in animal macrophages using mathematical models
• Delay in ATP-dependent calcium inflow may affect insulin secretion from pancreatic beta-cell
• Modeling basic aspects of bacterial resistance of Mycobacterium tuberculosis to antibiotics
• Mathematical model for the growth of mycobacterium tuberculosis in the granuloma
• Understanding PGE2, LXA4 and LTB4 balance during \textit{Mycobacterium tuberculosis} infection through mathematical model
• Calcium dynamics in cardiac excitatory and non-excitatory cells and the role of gap junction
• Requirement for multiple activation signals by anti-inflammatory feedback in macrophages
• Mathematical analysis of macrophage-bacteria interaction in tuberculosis infection
• Dynamic mathematical model development and validation of \textit{in vitro Mycobacterium smegmatis} growth under nutrient- and pH-stress
• Modeling innate immune response to early Mycobacterium infection
• Restoration of cytosolic calcium inhibits \textit{Mycobacterium tuberculosis} intracellular growth: theoretical evidence and experimental observation
• Modeling the macrophage-anthrax spore interaction: implications for early host-pathogen interactions

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