Two-stage model of carcinogenic mutations with the influence of delays

DOI10.3934/DCDSB.2014.19.2501zbMATH Open1304.35734OpenAlexW2326087378MaRDI QIDQ478737FDOQ478737

Authors: Beata Zduniak, Urszula Foryś 

Publication date: 4 December 2014

Published in: Discrete and Continuous Dynamical Systems. Series B (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.3934/dcdsb.2014.19.2501

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zbMATH Keywords

diffusionstability switchesdelay equationscarcinogenic mutations

Mathematics Subject Classification ID

Medical applications (general) (92C50) Dynamical systems in biology (37N25) Stability theory of functional-differential equations (34K20) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Partial functional-differential equations (35R10) General biology and biomathematics (92B05) Biological rhythms and synchronization (92B25) Dynamical aspects of attractors and their bifurcations (37G35)

Cites Work

• Introduction to functional differential equations
• On Traveling Wave Solutions of Fisher's Equation in Two Spatial Dimensions
• A simple model of carcinogenic mutations with time delay and diffusion
• Title not available (Why is that?)
• BIOLOGICAL DELAY SYSTEMS AND THE MIKHAILOV CRITERION OF STABILITY
• Multi-dimensional Lotka-Volterra systems for carcinogenesis mutations
• Oscillations and bistability in the stochastic model of p53 regulation
• Title not available (Why is that?)
• Stability Analysis and Comparison of the Models for Carcinogenesis Mutations in the Case of Two Stages of Mutations

Cited In (7)

• A waiting time problem arising from the study of multi-stage carcinogenesis
• Two-mutation model for carcinogenesis: Joint analysis of premalignant and malignant lesions
• A simple model of carcinogenic mutations with time delay and diffusion
• Stability Analysis and Comparison of the Models for Carcinogenesis Mutations in the Case of Two Stages of Mutations
• The Dynamic Model for Cancer Relapse Based on Two-Stage Model of Carcinogenesis
• Stability and optimal control of a delayed HIV/AIDS-PrEP model
• Are Two Mutations Sufficient to Cause Cancer? Some Generalizations of the Two-Mutation Model of Carcinogenesis of Moolgavkar, Venzon, and Knudson, and of the Multistage Model of Armitage and Doll

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