The response to antiangiogenic anticancer drugs that inhibit endothelial cell proliferation
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Publication:856106
DOI10.1016/j.amc.2006.01.061zbMath1101.92026OpenAlexW2026154594MaRDI QIDQ856106
Alberto d'Onofrio, Alberto Gandolfi
Publication date: 7 December 2006
Published in: Applied Mathematics and Computation (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.amc.2006.01.061
Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60) Global stability of solutions to ordinary differential equations (34D23)
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Cites Work
- Periodic motions
- Globally stable vaccine-induced eradication of horizontally and vertically transmitted infectious diseases with periodic contact rates and disease-dependent demographic factors in the population
- Stability properties of pulse vaccination strategy in SEIR epidemic model
- Tumour eradication by antiangiogenic therapy: analysis and extensions of the model by Hahnfeldt et al. (1999)
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