Effects of CCN1 and macrophage content on glioma virotherapy: a mathematical model

DOI10.1007/S11538-015-0074-8zbMATH Open1335.92040OpenAlexW2050110755WikidataQ42155868 ScholiaQ42155868MaRDI QIDQ891840FDOQ891840

Authors Luke Russell, Balveen Kaur, A. Friedman, Karly Jacobsen

Publication date 17 November 2015

Published in Bulletin of Mathematical Biology (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1007/s11538-015-0074-8

zbMATH Keywords

oncolytic virusanti-tumorccn1macrophage content

Mathematics Subject Classification ID

Medical applications (general) (92C50) Medical epidemiology (92C60)

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Cites work

• A methodology for performing global uncertainty and sensitivity analysis in systems biology
• Hypoxia inducible factors-mediated inhibition of cancer by GM-CSF: a mathematical model
• Interaction of tumor with its micro-environment: a mathematical model
• Time dependent problems and difference methods

Cited in

(11)

• Natural killer cells recruitment in oncolytic virotherapy: a mathematical model
• Multiscale modelling of cancer response to oncolytic viral therapy
• Complex role of NK cells in regulation of oncolytic virus-bortezomib therapy
• A critical virus production rate for efficiency of oncolytic virotherapy
• Computational modeling of cancer response to oncolytic virotherapy: improving the effectiveness of viral spread and anti tumor efficacy
• Increase hemoglobin level in severe malarial anemia while controlling parasitemia: a mathematical model
• Mathematical modeling of glioma therapy using oncolytic viruses
• Treatment of glioma with virotherapy and TNF-\(\alpha\) inhibitors: analysis as a dynamical system
• Non-local multiscale approaches for tumour-oncolytic viruses interactions
• The role of TNF-\(\alpha\) inhibitor in glioma virotherapy: a mathematical model
• Boundedness of the solution to a higher-dimensional triply haptotactic cross-diffusion system modeling oncolytic virotherapy

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