Hypoxia inducible factors-mediated inhibition of cancer by GM-CSF: a mathematical model
DOI10.1007/S11538-012-9776-3zbMATH Open1267.92043OpenAlexW1997851555WikidataQ37606594 ScholiaQ37606594MaRDI QIDQ1936712FDOQ1936712
Authors: Duan Chen, Julie M. Roda, Clay B. Marsh, Timothy D. Eubank, A. Friedman 
Publication date: 6 February 2013
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: http://europepmc.org/articles/pmc3936579
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Medical applications (general) (92C50) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Free boundary problems for PDEs (35R35)
Cites Work
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- A methodology for performing global uncertainty and sensitivity analysis in systems biology
- A model of wound-healing angiogenesis in soft tissue
- Modeling the inhibition of breast cancer growth by GM-CSF
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- The migration of cells in multicell tumor spheroids
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Cited In (18)
- On a nonlinear model for tumor growth in a cellular medium
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- Overcoming drug resistance to BRAF inhibitor
- Analysis and simulations on a model for the evolution of tumors under the influence of nutrient and drug application
- Mathematical modeling and analysis of CD200-CD200R in cancer treatment
- Modeling the inhibition of breast cancer growth by GM-CSF
- On a nonlinear model for tumor growth: global in time weak solutions
- Mathematical modeling of cellular cross-talk between endothelial and tumor cells highlights counterintuitive effects of VEGF-targeted therapies
- The role of the cytokines IL-27 and IL-35 in cancer
- Deterministic and stochastic aspects of VEGF-A production and the cooperative behavior of tumoral cell colony
- The role of CD200-CD200R in tumor immune evasion
- Effects of CCN1 and macrophage content on glioma virotherapy: a mathematical model
- Modeling hypoxia-related inflammation scenarios
- Spatio-temporal model of combining chemotherapy with senolytic treatment in lung cancer
- A two-phase free boundary problem with discontinuous velocity: Application to tumor model
- A mathematical model of HiF-\(1{\alpha}\)-mediated response to hypoxia on the G1/S transition
- Control of tumor growth modeled by system of PDE, numerical analysis of optimality conditions
- Mathematical modeling in scheduling cancer treatment with combination of VEGF inhibitor and chemotherapy drugs
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