Mathematical modeling and analysis of CD200-CD200R in cancer treatment

DOI10.1007/S11538-022-01039-XMaRDI QIDQ2154530zbMATH OpenOpenAlexWikidataFDO

Authors Kang-Ling Liao, Kenton D. Watt

Publication date 19 July 2022

Published in Bulletin of Mathematical Biology (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1007/s11538-022-01039-x

zbMATH Keywords

populationbifurcation analysisbinding affinityODE modelsCD200-CD200R

Mathematics Subject Classification ID

Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60) Cell biology (92C37)

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Cites work

• A kinetic theory approach for modelling tumour and macrophages heterogeneity and plasticity during cancer progression
• A mathematical model for pancreatic cancer growth and treatments
• A mathematical model of tumour angiogenesis, regulated by vascular endothelial growth factor and the angiopoietins
• A methodology for performing global uncertainty and sensitivity analysis in systems biology
• Hypoxia inducible factors-mediated inhibition of cancer by GM-CSF: a mathematical model
• Mathematical modeling in scheduling cancer treatment with combination of VEGF inhibitor and chemotherapy drugs
• Modeling the inhibition of breast cancer growth by GM-CSF
• Modelling the interactions between tumour cells and a blood vessel in a microenvironment within a vascular tumour
• Oncolytic viral therapies and the delicate balance between virus-macrophage-tumour interactions: a mathematical approach
• The human immune response to \textit{Mycobacterium tuberculosis} in lung and lymph node
• The re-polarisation of M2 and M1 macrophages and its role on cancer outcomes
• The role of CD200-CD200R in tumor immune evasion

Cited in

(3)

• Mathematical modeling of mutant transferrin-CRM107 molecular conjugates for cancer therapy
• Different mechanisms of CD200-CD200R induce diverse outcomes in cancer treatment
• The role of CD200-CD200R in tumor immune evasion

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