The re-polarisation of M2 and M1 macrophages and its role on cancer outcomes
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Cites work
- A mathematical model for pancreatic cancer growth and treatments
- A mathematical model for the immunotherapeutic control of the Th1/Th2 imbalance in melanoma
- Mathematical modeling of allergy and specific immunotherapy: Th1-Th2-Treg interactions
- Modeling anti-tumor Th1 and Th2 immunity in the rejection of melanoma
- Regulation of Th1/Th2 cells in asthma development: a mathematical model
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(19)- A kinetic theory approach for modelling tumour and macrophages heterogeneity and plasticity during cancer progression
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- Non-local parabolic and hyperbolic models for cell polarisation in heterogeneous cancer cell populations
- Mathematical modeling and bifurcation analysis of pro- and anti-tumor macrophages
- A detailed study on a tumor model with delayed growth of pro-tumor macrophages
- A mathematical model for the immune-mediated theory of metastasis
- Implications of immune-mediated metastatic growth on metastatic dormancy, blow-up, early detection, and treatment
- Modeling the dichotomy of the immune response to cancer: cytotoxic effects and tumor-promoting inflammation
- A structural methodology for modeling immune-tumor interactions including pro- and anti-tumor factors for clinical applications
- Investigation into the role of macrophages heterogeneity on solid tumour aggregations
- A mathematical model for the role of macrophages in the persistence and elimination of oncolytic viruses
- Mathematical investigation of the role of re-polarisation of M2 in cancer therapy
- An investigation on Michaelis-Menten kinetics based complex dynamics of tumor-immune interaction
- Algorithmic asymptotic analysis: extending the arsenal of cancer immunology modeling
- Mathematical investigation into the role of macrophage heterogeneity on the temporal and spatio-temporal dynamics of non-small cell lung cancers
- A mathematical study of the role of tbregs in breast cancer
- Modelling and investigation of the \(\mathrm{CD}4^+\) T cells -- macrophages paradox in melanoma immunotherapies
- Mathematical modeling of tumor-immune cell interactions
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