Modelling cell migration strategies in the extracellular matrix
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Publication:2339992
DOI10.1007/S00285-008-0217-8zbMATH Open1311.92041OpenAlexW2169659086WikidataQ38454449 ScholiaQ38454449MaRDI QIDQ2339992FDOQ2339992
Authors: Kevin J. Painter 
Publication date: 15 April 2015
Published in: Journal of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s00285-008-0217-8
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Cited In (49)
- The diffusion limit of transport equations in biology
- Mathematical modeling of cancer cell invasion of tissue: biological insight from mathematical analysis and computational simulation
- An integro-differential non-local model for cell migration and its efficient numerical solution
- Biased three-dimensional cell migration and collagen matrix modification
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- Phenotypic switching mechanisms determine the structure of cell migration into extracellular matrix under the `go-or-grow' hypothesis
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- Multi-cue kinetic model with non-local sensing for cell migration on a fiber network with chemotaxis
- Modeling of adhesion, protrusion, and contraction coordination for cell migration simulations
- A positivity preserving adaptive moving mesh method for cancer cell invasion models
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- On the shape of invading population in anisotropic environments
- Semi-autonomous wound invasion via matrix-deposited, haptotactic cues
- Anisotropic diffusion in oriented environments can lead to singularity formation
- 3D computational modelling of cell migration: a mechano-chemo-thermo-electrotaxis approach
- A mathematical model for mesenchymal and chemosensitive cell dynamics
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- Traveling wave solutions for a cancer stem cell invasion model
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- Continuum model of cell motility and chemotaxis
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- Direction-dependent turning leads to anisotropic diffusion and persistence
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- Cell-scale degradation of peritumoural extracellular matrix fibre network and its role within tissue-scale cancer invasion
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- Multiscale dynamics of a heterotypic cancer cell population within a fibrous extracellular matrix
- A new model for the emergence of blood capillary networks
- Soluble VEGFR1 signaling guides vascular patterns into dense branching morphologies
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