MATHEMATICAL MODELLING OF CANCER CELL INVASION OF TISSUE: THE ROLE OF THE UROKINASE PLASMINOGEN ACTIVATION SYSTEM
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Cited in
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- Boundedness in a three-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- Numerical solutions for a model of tissue invasion and migration of tumour cells
- Evolution of cell motility in an individual-based model of tumour growth
- Mathematical modelling of cancer invasion: the multiple roles of TGF-\(\beta\) pathway on tumour proliferation and cell adhesion
- Multiscale modelling of fibres dynamics and cell adhesion within moving boundary cancer invasion
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- Spatio-temporal oscillations in the Keller-Segel system with logistic growth
- Large time behavior of solution to a fully parabolic chemotaxis-haptotaxis model in higher dimensions
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- Chemotaxis-induced spatio-temporal heterogeneity in multi-species host-parasitoid systems
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- Toward a mathematical theory of Keller-Segel models of pattern formation in biological tissues
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- Global existence of a two-dimensional chemotaxis-haptotaxis model with remodeling of non-diffusible attractant
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- Global existence of classical solutions to a combined chemotaxis-haptotaxis model with logistic source
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- Directionality of macrophages movement in tumour invasion: a multiscale moving-boundary approach
- Boundedness of solutions to a quasilinear higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- Existence and uniqueness of global classical solutions to a two dimensional two species cancer invasion haptotaxis model
- Mathematical modelling of cancer invasion of tissue: dynamic heterogeneity
- MATHEMATICAL MODELLING OF CANCER INVASION OF TISSUE: THE ROLE AND EFFECT OF NONLOCAL INTERACTIONS
- Optimal control and pattern formation for a haptotaxis model of solid tumor invasion
- Computational approaches and analysis for a spatio-structural-temporal invasive carcinoma model
- Galerkin finite element method for cancer invasion mathematical model
- Large-data solutions in a three-dimensional chemotaxis-haptotaxis system with remodeling of non-diffusible attractant: the role of sub-linear production of diffusible signal
- Emergence of large population densities despite logistic growth restrictions in fully parabolic chemotaxis systems
- Solvability of solid tumor invasion model
- A positivity preserving adaptive moving mesh method for cancer cell invasion models
- Mathematical analysis and numerical simulations for the cancer tissue invasion model
- Global boundedness of solutions to a chemotaxis-haptotaxis model with tissue remodeling
- Chemotactic aggregation versus logistic damping on boundedness in the 3D minimal Keller-Segel model
- On a bi-dimensional chemo-repulsion model with nonlinear production and a related optimal control problem
- Two positivity preserving flux limited, second-order numerical methods for a haptotaxis model
- Numerical experiments with model equations of cancer invasion of tissue
- Critical mass for infinite-time blow-up in a haptotaxis system with nonlinear zero-order interaction
- Multi-parametric evolution of conditions leading to cancer invasion in biological systems
- Possible points of blow-up in chemotaxis systems with spatially heterogeneous logistic source
- Modeling multiple taxis: tumor invasion with phenotypic heterogeneity, haptotaxis, and unilateral interspecies repellence
- Finite-time blow-up in low-dimensional Keller-Segel systems with logistic-type superlinear degradation
- A novel 3D atomistic-continuum cancer invasion model: in silico simulations of an \textit{in vitro} organotypic invasion assay
- Structured models of cell migration incorporating molecular binding processes
- Splitting Schemes and Segregation in Reaction Cross-Diffusion Systems
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- On a distributed control problem for a coupled chemotaxis-fluid model
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