MATHEMATICAL MODELLING OF CANCER CELL INVASION OF TISSUE: THE ROLE OF THE UROKINASE PLASMINOGEN ACTIVATION SYSTEM
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Publication:5715596
DOI10.1142/S0218202505000947zbMATH Open1094.92039OpenAlexW2081615148WikidataQ111491175 ScholiaQ111491175MaRDI QIDQ5715596FDOQ5715596
Authors: Georgios Lolas, M. A. J. Chaplain 
Publication date: 4 January 2006
Published in: M\(^3\)AS. Mathematical Models \& Methods in Applied Sciences (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1142/s0218202505000947
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Medical applications (general) (92C50) Reaction-diffusion equations (35K57) Cell movement (chemotaxis, etc.) (92C17)
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Cited In (only showing first 100 items - show all)
- On the boundedness and decay of solutions for a chemotaxis-haptotaxis system with nonlinear diffusion
- Mathematical modelling of cancer invasion: a review
- BOUNDEDNESS OF SOLUTIONS OF A HAPTOTAXIS MODEL
- FROM THE PHYSICAL LAWS OF TUMOR GROWTH TO MODELLING CANCER PROCESSES
- How far can chemotactic cross-diffusion enforce exceeding carrying capacities?
- PREFACE — CHALLENGING MATHEMATICAL PROBLEMS IN CANCER MODELLING
- Spatio-temporal chaos in a chemotaxis model
- Mathematical modeling of cancer cell invasion of tissue: biological insight from mathematical analysis and computational simulation
- LOOKING FOR NEW PARADIGMS TOWARDS A BIOLOGICAL-MATHEMATICAL THEORY OF COMPLEX MULTICELLULAR SYSTEMS
- Existence and uniqueness of global classical solutions to a two dimensional two species cancer invasion haptotaxis model
- Mathematical modelling of cancer invasion of tissue: dynamic heterogeneity
- Local existence and uniqueness of regular solutions in a model of tissue invasion by solid tumours
- Global existence of classical solutions to a combined chemotaxis-haptotaxis model with logistic source
- Multiscale modelling of cancer response to oncolytic viral therapy
- Boundedness in a multi-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- A mathematical insight in the epithelial-mesenchymal-like transition in cancer cells and its effect in the invasion of the extracellular matrix
- Adaptive grid modelling for cancer cells in the early stage of invasion
- A note on the numerical approach for the reaction-diffusion problem to model the density of the tumor growth dynamics
- Toward a mathematical theory of Keller–Segel models of pattern formation in biological tissues
- Critical mass for infinite-time aggregation in a chemotaxis model with indirect signal production
- Segregation effects and gap formation in cross-diffusion models
- Mathematical modeling and numerical simulation of a multiscale cancer invasion of host tissue
- Finite-time blow-up of solutions to a cancer invasion mathematical model with haptotaxis effects
- An adaptive rectangular mesh administration and refinement technique with application in cancer invasion models
- Mathematical modelling of cancer invasion: implications of cell adhesion variability for tumour infiltrative growth patterns
- Boundedness of solutions to a quasilinear higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- Boundedness in a three-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- An element-free Galerkin meshless method for simulating the behavior of cancer cell invasion of surrounding tissue
- A positivity preserving adaptive moving mesh method for cancer cell invasion models
- Mathematical tools of the kinetic theory of active particles with some reasoning on the modelling progression and heterogeneity
- Asymptotic stability in a quasilinear chemotaxis-haptotaxis model with general logistic source and nonlinear signal production
- Global existence of classical solutions to a chemotaxis-haptotaxis model
- Boundedness in a quasilinear chemotaxis-haptotaxis system with logistic source
- Large time behavior of solution to a fully parabolic chemotaxis-haptotaxis model in higher dimensions
- Boundedness in the higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- From the mathematical kinetic theory of active particles to multiscale modelling of complex biological systems
- Hopf bifurcation in a solid avascular tumour growth model with two discrete delays
- STOCHASTIC MODELING OF LOSS- AND GAIN-OF-FUNCTION MUTATIONS IN CANCER
- A novel method for simulating the extracellular matrix in models of tumour growth
- Boundedness of solutions of a non-local reaction–diffusion model for adhesion in cell aggregation and cancer invasion
- Large Time Behavior in a Multidimensional Chemotaxis-Haptotaxis Model with Slow Signal Diffusion
- Finite time blow-up in the higher dimensional parabolic-elliptic-ODE minimal chemotaxis-haptotaxis system
- Modelling Aspects of Cancer Growth: Insight from Mathematical and Numerical Analysis and Computational Simulation
- Numerical solutions for a model of tissue invasion and migration of tumour cells
- NUMERICAL VERSUS EXPERIMENTAL DATA FOR PROSTATE TUMOUR GROWTH
- On a parabolic-elliptic chemotactic model with coupled boundary conditions
- Nonlinear simulations of solid tumor growth using a mixture model: invasion and branching
- Global boundedness and decay for a multi-dimensional chemotaxis-haptotaxis system with nonlinear diffusion
- A mathematical model of breast cancer development, local treatment and recurrence
- Boundedness and asymptotic behavior of solutions to a chemotaxis-haptotaxis model in high dimensions
- Mathematical modelling of cancer cell invasion of tissue: local and non-local models and the effect of adhesion
- Mathematical modelling of cancer cell invasion of tissue
- A Hybrid Multiscale Model for Cancer Invasion of the Extracellular Matrix
- Parabolic equations with dynamical boundary conditions and source terms on interfaces
- Global asymptotic stability of constant equilibria in a fully parabolic chemotaxis system with strong logistic dampening
- $L^1$ solutions to parabolic Keller-Segel systems involving arbitrary superlinear degradation
- Boundedness of solutions to a quasilinear chemotaxis-haptotaxis model
- Spatio-temporal oscillations in the Keller–Segel system with logistic growth
- Asymptotic behavior of global solutions to a model of cell invasion
- Evolution of cell motility in an individual-based model of tumour growth
- Mathematical modelling of the influence of heat shock proteins on cancer invasion of tissue
- Mathematical modelling of cancer invasion: the importance of cell -- cell adhesion and cell -- matrix adhesion
- A mathematical framework for modelling the metastatic spread of cancer
- A free boundary problem modeling the cell cycle and cell movement in multicellular tumor spheroids
- MATHEMATICAL MODELLING OF CANCER INVASION OF TISSUE: THE ROLE AND EFFECT OF NONLOCAL INTERACTIONS
- Modelling cell migration strategies in the extracellular matrix
- Large-Data Global Generalized Solutions in a Chemotaxis System with Tensor-Valued Sensitivities
- Directionality of macrophages movement in tumour invasion: a multiscale moving-boundary approach
- Microenvironment driven invasion: a multiscale multimodel investigation
- Multiscale agent-based cancer modeling
- Loss- and gain-of-function mutations in cancer: mass-action, spatial and hierarchical models
- Chemotaxis-induced spatio-temporal heterogeneity in multi-species host-parasitoid systems
- Slow and fast invasion waves in a model of acid-mediated tumour growth
- Global weak solutions in a three-dimensional two-species cancer invasion haptotaxis model without cell proliferation
- A study on time discretization and adaptive mesh refinement methods for the simulation of cancer invasion: the urokinase model
- The impact of adhesion on cellular invasion processes in cancer and development
- Multiscale modelling of fibres dynamics and cell adhesion within moving boundary cancer invasion
- A multiscale mathematical model of tumour invasive growth
- CONVERGENCE OF A CANCER INVASION MODEL TO A LOGISTIC CHEMOTAXIS MODEL
- Space-time adaptivity for a multi-scale cancer invasion model
- Global existence of a two-dimensional chemotaxis-haptotaxis model with remodeling of non-diffusible attractant
- Existence of weak solutions to the Keller-Segel chemotaxis system with additional cross-diffusion
- Attractiveness of constant states in logistic-type Keller-Segel systems involving subquadratic growth restrictions
- A new result for 2D boundedness of solutions to a chemotaxis–haptotaxis model with/without sub-logistic source
- Chemotactic cross-diffusion in complex frameworks
- A Stokes Two-Fluid Model for Cell Migration that Can Account for Physical Cues in the Microenvironment
- On a bi-dimensional chemo-repulsion model with nonlinear production and a related optimal control problem
- Galerkin finite element method for cancer invasion mathematical model
- Finite-time blow-up in low-dimensional Keller-Segel systems with logistic-type superlinear degradation
- A new (and optimal) result for the boundedness of a solution of a quasilinear chemotaxis-haptotaxis model (with a logistic source)
- Global classical solutions and convergence to a mathematical model for cancer cells invasion and metastatic spread
- Global boundedness in a three-dimensional chemotaxis-haptotaxis model
- Local and nonlocal phase-field models of tumor growth and invasion due to ECM degradation
- On a distributed control problem for a coupled chemotaxis-fluid model
- A quasilinear chemotaxis-haptotaxis model: The roles of nonlinear diffusion and logistic source
- Large-data solutions in a three-dimensional chemotaxis-haptotaxis system with remodeling of non-diffusible attractant: the role of sub-linear production of diffusible signal
- A chemotaxis-haptotaxis system with haptoattractant remodeling: boundedness enforced by mild saturation of signal production
- Large time behavior of solutions to a fully parabolic chemotaxis-haptotaxis model in \(N\) dimensions
- Global existence for an attraction-repulsion chemotaxis fluid model with logistic source
- Solvability of solid tumor invasion model
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