Modeling the spatial distribution of chronic tumor hypoxia: implications for experimental and clinical studies
DOI10.1155/2012/410602zbMATH Open1233.92037DBLPjournals/cmmm/GibinKMS12OpenAlexW2044784870WikidataQ41573454 ScholiaQ41573454MaRDI QIDQ764161FDOQ764161
Authors: Gibin G. Powathil, Mohammad Kohandel, Michael Milosevic, S. Sivaloganathan
Publication date: 13 March 2012
Published in: Computational \& Mathematical Methods in Medicine (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1155/2012/410602
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Cited In (13)
- A mathematical model of tumor hypoxia targeting in cancer treatment and its numerical simulation
- Targeting cellular DNA damage responses in cancer: an in vitro-calibrated agent-based model simulating monolayer and spheroid treatment responses to ATR-inhibiting drugs
- Towards the virtual tumor for optimizing radiotherapy treatments of hypoxic tumors: a novel model of heterogeneous tissue vasculature and oxygenation
- Investigating the influence of growth arrest mechanisms on tumour responses to radiotherapy
- Transient alleviation of tumor hypoxia during first days of antiangiogenic therapy as a result of therapy-induced alterations in nutrient supply and tumor metabolism -- analysis by mathematical modeling
- Bystander effects and their implications for clinical radiation therapy: insights from multiscale \textit{in silico} experiments
- Computational study of the effect of hypoxia on cancer response to radiation treatment
- Phenotypic variation modulates the growth dynamics and response to radiotherapy of solid tumours under normoxia and hypoxia
- Modeling the emergence of phenotypic heterogeneity in vascularized tumors
- Assessing the therapeutic response of tumors to hypoxia-targeted prodrugs with an \textit{in silico} approach
- Modelling the effects of cell-cycle heterogeneity on the response of a solid tumour to chemotherapy: biological insights from a hybrid multiscale cellular automaton model
- Modelling tumour oxygenation, reoxygenation and implications on treatment outcome
- What does not kill a tumour may make it stronger: \textit{in silico} insights into chemotherapeutic drug resistance
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