HIV dynamics: analysis and robust multirate MPC-based treatment schedules
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Publication:2272049
DOI10.1016/J.JMAA.2009.05.038zbMATH Open1166.92025OpenAlexW2132301640MaRDI QIDQ2272049FDOQ2272049
Publication date: 5 August 2009
Published in: Journal of Mathematical Analysis and Applications (Search for Journal in Brave)
Full work available at URL: http://hdl.handle.net/2263/11279
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Cited In (19)
- Global properties of a class of HIV infection models with Beddington–DeAngelis functional response
- Title not available (Why is that?)
- Global dynamics of HIV infection of CD4 T cells and macrophages
- Kinetic model of HIV infection including hematopoietic progenitor cells
- Global properties of a cell mediated immunity in HIV infection model with two classes of target cells and distributed delays
- Global dynamics of an HIV infection model with two classes of target cells and distributed delays
- Stability and feedback stabilization of HIV infection model with two classes of target cells
- Global properties of a class of virus infection models with multitarget cells
- Stability analysis for viral infection model with multitarget cells, Beddington-DeAngelis functional response, and humoral immunity
- A singularly perturbed vector‐bias malaria model incorporating bed net control
- Hepatitis B virus dynamics: modeling, analysis, and optimal treatment scheduling
- Dynamical behaviors of a general humoral immunity viral infection model with distributed invasion and production
- Global properties of delayed-HIV dynamics models with differential drug efficacy in cocirculating target cells
- Global properties of a class of HIV models
- Global stability of a delayed virus dynamics model with multi-staged infected progression and humoral immunity
- Global dynamics of delay‐distributed HIV infection models with differential drug efficacy in cocirculating target cells
- Global stability of HIV infection of CD4\(^{+}\) T cells and macrophages with CTL immune response and distributed delays
- Global properties of virus dynamics models with multitarget cells and discrete-time delays
- Stability of a delayed SARS-CoV-2 reactivation model with logistic growth and adaptive immune response
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