Simple ODE models of tumor growth and anti-angiogenic or radiation treatment

From MaRDI portal
Publication:5950554


DOI10.1016/S0895-7177(00)00316-2zbMath1004.92023MaRDI QIDQ5950554

Philip Hahnfeldt, Rainer K. Sachs, Lynn R. Hlatky

Publication date: 2 January 2002

Published in: Mathematical and Computer Modelling (Search for Journal in Brave)


37N25: Dynamical systems in biology

92C50: Medical applications (general)


Related Items

Nonautonomous Dynamical Systems in the Life Sciences, Periodic solutions of angiogenesis models with time lags, Observer-based techniques for the identification and analysis of avascular tumor growth, Inference on a stochastic two-compartment model in tumor growth, Angiogenesis inhibition and tumor-immune interactions with chemotherapy by a control set-valued method, Analysis of the Hopf bifurcation for the family of angiogenesis models, Analysis of the Hopf bifurcation for the family of angiogenesis models. II: The case of two nonzero unequal delays, Estimating tumor growth rates in vivo, Stability criteria for the cure state in a cancer model with radiation treatment, Mathematical modelling of tissue-engineered angiogenesis, On optimal delivery of combination therapy for tumors, Tumour eradication by antiangiogenic therapy: analysis and extensions of the model by Hahnfeldt et al. (1999), Assessing the effects of estrogen on the dynamics of breast cancer, Stability of Hahnfeldt angiogenesis models with time lags, A mathematical model for brain tumor response to radiation therapy, Linear quadratic and tumour control probability modelling in external beam radiotherapy, Effect of treatment on the global dynamics of delayed pathological angiogenesis models, Persistence and extinction in a mathematical model of cell populations affected by radiation, A bi-parametric model for the tumour angiogenesis and antiangiogenesis therapy, Interactions between the immune system and cancer: A brief review of non-spatial mathematical models, Mesoscopic and continuum modelling of angiogenesis, Optimal control of the dynamics of a tumor growth model with Hollings' type-II functional response



Cites Work