Optimal control of a phase field tumor growth model with chemotaxis and active transport
From MaRDI portal
Publication:6494300
DOI10.23952/JNVA.8.2024.1.03MaRDI QIDQ6494300
Publication date: 30 April 2024
Published in: Journal of Nonlinear and Variational Analysis (Search for Journal in Brave)
Reaction-diffusion equations (35K57) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Medical applications (general) (92C50) Cell movement (chemotaxis, etc.) (92C17)
Cites Work
- Unnamed Item
- Unnamed Item
- Unnamed Item
- On a Cahn-Hilliard type phase field system related to tumor growth
- Well-posedness and global attractor of the Cahn-Hilliard-Brinkman system with dynamic boundary conditions
- Analysis of a Cahn-Hilliard system with non-zero Dirichlet conditions modeling tumor growth with chemotaxis
- On the Cahn-Hilliard-Brinkman system
- Finite-dimensional global attractor of the Cahn-Hilliard-Brinkman system
- Vanishing viscosities and error estimate for a Cahn-Hilliard type phase field system related to tumor growth
- Infinite-dimensional dynamical systems in mechanics and physics.
- Optimal control of treatment time in a diffuse interface model of tumor growth
- Analysis of a Cahn-Hilliard-Brinkman model for tumour growth with chemotaxis
- Three-dimensional multispecies nonlinear tumor growth. I: Model and numerical method
- Optimal medication for tumors modeled by a Cahn-Hilliard-Brinkman equation
- Long-time dynamics and optimal control of a diffuse interface model for tumor growth
- Penalisation of long treatment time and optimal control of a tumour growth model of Cahn-Hilliard type with singular potential
- Optimal control of a phase field system modelling tumor growth with chemotaxis and singular potentials
- Long-time dynamics for a Cahn-Hilliard tumor growth model with chemotaxis
- Optimality conditions for an extended tumor growth model with double obstacle potential via deep quench approach
- Global weak solutions and asymptotic limits of a Cahn-Hilliard-Darcy system modelling tumour growth
- Nonlinear simulations of solid tumor growth using a mixture model: invasion and branching
- Well-posedness and long-time behavior of a non-autonomous Cahn-Hilliard-Darcy system with mass source modeling tumor growth
- On the long time behavior of a tumor growth model
- Bayesian calibration, validation, and uncertainty quantification of diffuse interface models of tumor growth
- Optimal control problems with sparsity for tumor growth models involving variational inequalities
- Optimal Distributed Control of a Nonlocal Cahn--Hilliard/Navier--Stokes System in Two Dimensions
- A Cahn–Hilliard–Darcy model for tumour growth with chemotaxis and active transport
- Analysis of a mixture model of tumor growth
- Numerical simulation of a thermodynamically consistent four-species tumor growth model
- Second-order analysis of an optimal control problem in a phase field tumor growth model with singular potentials and chemotaxis
- GENERAL DIFFUSE-INTERFACE THEORIES AND AN APPROACH TO PREDICTIVE TUMOR GROWTH MODELING
- On a diffuse interface model of tumour growth
- Well-posedness of a Cahn–Hilliard system modelling tumour growth with chemotaxis and active transport
- Exponential attractors for the Cahn-Hilliard equation with dynamic boundary conditions
- Sparse optimal control of a phase field system with singular potentials arising in the modeling of tumor growth
- Existence of weak solutions to multiphase Cahn–Hilliard–Darcy and Cahn–Hilliard–Brinkman models for stratified tumor growth with chemotaxis and general source terms
- Vanishing parameter for an optimal control problem modeling tumor growth
- Formal asymptotic limit of a diffuse-interface tumor-growth model
- Analysis of a diffuse interface model of multispecies tumor growth
- Optimal distributed control of a diffuse interface model of tumor growth
- Optimal Control of a Semidiscrete Cahn--Hilliard--Navier--Stokes System
- Sparse Optimal Control of a Phase Field Tumor Model with Mechanical Effects
This page was built for publication: Optimal control of a phase field tumor growth model with chemotaxis and active transport